Ldl Cholesterol
How To Lower LDL Cholesterol (Bad Cholesterol)
For years how to lower LDL cholesterol was not a subject that was even discussed much in medical circles. Up until the late seventies or early eighties it wouldn’t be unusual to find your doctor smoking 2 packs of cigarettes a day and a diet that consisted of eggs and sausage for breakfast with a chili cheese dog thrown in for good measure for lunch. But since that time things have change dramatically, perhaps due to the realization that heart disease is Americas number one killer ahead of every type of cancer combined and every infectious and degenerative disease.
LDL cholesterol (low density lipoproteins) are one of the major contributing factors to heart and artery disease, and finding out how to lower LDL cholesterol could be the first step in avoiding a major medical calamity.
In a small percentage of people high LDL levels are caused by a genetic condition called hypercholesterolemia. More commonly though, high bad Cholesterol Levels are attributed to other causes. In this article titled addressing how to lower LDL cholesterol we will be focusing on non hypercholesterolemia cuases.
Usually how to lower LDL cholesterol is addressed best by a handful of simple steps. These include losing weight (if needed), giving up the cigarettes (if a smoker), limited the amount of fat and cholesterol consumed, moderate exercise, and when necessary implement a statin or natural cholesterol supplement. Sounds pretty simple, but as we all know old habits can be hard to break.
Most experts agree that if how to lower LDL cholesterol is your focus you should consume no more than 30 percent of your total calories from fats. Saturated (harmful fats) should be limited to about 8 percent or less of total calories consumed per day. Some research suggests that by substituting polyunsaturated fats for saturated fats you may be able to substantially reduce bad cholesterol in the blood dramatically over a short period of time. While your head may be spinning after reading this paragraph don’t despair, simply read the label before you purchase those delicious all meat franks and bargain priced chili topping.
For those of you who follow my writing you likely know what is coming next; Exercise! Before you skip over this paragraph I promise to make it short. In the world of how to lower LDL cholesterol, exercise is an intricate component. Regular physical exercise can help lower LDL (bad cholesterol) and raise (HDL) by adopting a regime consisting of three 30 minute walks per week.
What Next? How to lower LDL cholesterol, in most cases, is about changing old habits, adopting new healthier habits, and enlisting the help of convention medications such as statins or natural cholesterol reducing remedies if needed. Put simply, this basically means finding ways to increase HDL (Good Cholesterol) and decreasing LDL (bad cholesterol levels). Certainly the aforementioned statin drugs will be one of your options but they do carry a number of serious label warnings. The side effect risk have made natural cholesterol reduction supplements containing such beneficial ingredients as lecithin oil, pumpkin seem oil, D-limonene, phytosterols, and antioxidants such as selenium, theaflavin, and tea catechins an alternative treatment option worth considering.
About the Author
Rob D. Hawkins is an enthusiastic advocate for the use of natural health products and natural living with over 10 years
experience in the field.
Learn more about natural remedies and natural health at
Purchase Remedies.com
Fat (LDL) Degradation: PMAP
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BMV Quantum Subliminal CD Lower Cholesterol: Low Cholesterol Mind Program (Ultrasonic Subliminal Series) $14.99 Program your subconscious mind to lower cholesterol levels. Create life-changing results using state-of-the-art subliminal and brainwave entrainment technologies. Tune your brainwaves to specific frequencies by listening to this CD! Program your subconscious mind for positive lasting results, created by a Certified Hypnotherapist and NLP Practitioner (Neuro-Linguistic Programming). Silent affirmat… |
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Dietary Management of Fat and Cholesterol (Video Counseling Library, Cardiology) This video explains how HDL, LDL, saturated fats, andunsaturated fats affect the body, and the importance ofreducing cholesterol levels. Viewers learn American Heart Association based diet strategies for healthier foods, howto read labels, and how to prepare foods…. |
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Heart Decisions For Life: Compliance (Cholesterol Watch) Discusses why compliance is so critical to regulating cholesterol. You must be an active participant in your own health to succeed in controlling your cholesterol…. |
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Cholesterol: Raising the Good and Lowering the Bad $9.99 … |
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Managing Cholesterol (Home Use) $24.95 Part of the award winning public television series Healthy Body/Healthy Mind. Heart disease is the number one killer of men and women in America and high cholesterol numbers are a major risk factor for both heart attacks and strokes. In this program we offer information about lifestyle changes that can help keep cholesterol numbers in the normal range, plus we see how changing the diet and adding… |
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Cholesterol: Raising the Good and Lowering the Bad (Home Use) $24.95 By now, most people have heard the term HDL – good cholesterol, and LDL – bad cholesterol. But do you know what makes good cholesterol good and bad cholesterol bad? And how does the wrong combination of these lipoproteins affect the risk of heart disease? This program will delve into these important questions and provide you with the answers you need to control your cholesterol.This product is man… |
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New Chapter Wholemega 1000mg NewExtra Virgin Omega-Rich Fish Oil100% Wild Alaskan SalmonNature’s Whole Complement of Sixteen Omegas – 3, 5, 6, 7 and 9’sBioavailable: Clinically Shown to Enter Cell Membranes for Optimal Fish Oil Benefits Effective: Reduces inflammatory Arachidonic Acid* Whole: Natural Source of Omegas + Vitamin D + Astaxanthin Heart HealthyThe Benefits of WholeWholemega™ is a true expression of nature’s w… |
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Benecol Smart Chews, Caramel, 120-Count Soft Chews $25.99 Benecol® Smart Chews ~ Proven to Reduce CholesterolTake twice a day to Reduce CholesterolFeel in control with Benecol® Smart Chews, a delicious and convenient way to lower cholesterol. Enjoy two to four great-tasting chews a day with meals and you will start to see results in as little as two weeks!About Benecol® Smart ChewsPlant Stanol Esters are derived from natural plant sources and found on… |
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Ginger Root By Nature’s Way 550 Mg Ginger Root by Nature’s Way 180 Capsules… |
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Himalaya Shuddha Guggulu Cholesterol Regulator 60 Capsules $0.01 The Archer Farms brand is known for its superior-quality, unique product offerings including a wide variety of gourmet groceries, appetizers and European-style baked goods…. |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $49.99 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $49.99 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $108 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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Atherosclerosis, Hypertension and Diabetes $178.25 This volume is devoted to atherosclerosis, hypertension, and diabetes, three of the most important disease conditions in the world today. Nutritional intervention, cholesterol lowering agents, lipids themselves, particularly oxidized LDL, protein modification by ADP-ribose, bone marrow study, endothelial cell dysfunction, angiotensin, and the role of infection and inflammation are all discussed in the context of atherosclerotic cardiovascular disease.The hypertension section focuses on factors that may be responsible for high blood pressure, such as genetic predisposition, vascular hyperplasia and remodeling, insulin resistance, neurological aspects such as hypothalamic peptides. Also discussed are the possible contributions of the cellular function of the endothelium, nutrition, kidney dysfunction, leptin, and the brain. Novel routes of drug delivery for treatment of hypertension is also a focus.As for diabetes, the risk factors and mechanisms responsible for diabetic vascular and cardiac dysfunction are discussed. Lipid profile changes and fibrinolysis in diabetic patients is detailed, along with adipogenesis, diabetic cardiomyopathy, energy metabolism in the diabetic heart, vanadate as an alternative to insulin, insulin resistance mechanisms, and neurotransmitters as targets for the prevention of cardiovascular disease and diabetes.These manuscripts were invited from scientists who presented state of the art lectures at the XVII World Congress of the International Society for Heart Research held in Winnipeg in July 2001. |
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Biopsychosocial outcomes of a resilience and diabetes self-management education intervention in African American adults with type 2 diabetes. $49.99 Type 2 diabetes (T2DM) currently affects more than three million African American adults with double the number expected by 2025. The most effective and safest treatment for T2DM is lifestyle change therapy, including healthful eating, monitoring of blood glucose, and physical activity. However, current lifestyle change interventions are limited in their scope to alter the behaviors of individuals to more healthful ones. These limitations may be attributed, in part, to a lack of attention given to enhancing an individual’s psychosocial process variables, such as resilience, coping skills, self-leadership, and empowerment. Incorporating resilience education into lifestyle change therapies is a novel approach that addresses the behavior modification limitations of current interventions by aiming to enhance psychosocial process variables. Therefore, the purpose of this project was to conduct a six-month pilot study to determine the feasibility of our resilience and diabetes self-management intervention, The Diabetes Coaching Program: Transforming Lives Through Resilience Education, in a convenience sample of African American adults (n=16) with T2DM. The intervention included four weekly resilience and diabetes education classes and eight bi-weekly support group sessions. Survey data and blood samples were collected at baseline and at six months. Twelve participants completed the study (75% retention). Results indicated that higher perceived stress scores were associated with less resilience, fewer adaptive coping skills, lower self-leadership, lower diabetes empowerment and greater depressive symptoms. However, diabetes empowerment was the only psychosocial process variable to be significantly enhanced by the intervention at six months. Weight, BMI, HbA1c, total cholesterol, LDL cholesterol, blood pressure, and IGF-1 levels were significantly decreased at six months, whereas, lymphocyte proliferation and physical activity were significantly increased. These data |
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Biopsychosocial outcomes of a resilience and diabetes self-management education intervention in African American adults with type 2 diabetes. $49.99 Type 2 diabetes (T2DM) currently affects more than three million African American adults with double the number expected by 2025. The most effective and safest treatment for T2DM is lifestyle change therapy, including healthful eating, monitoring of blood glucose, and physical activity. However, current lifestyle change interventions are limited in their scope to alter the behaviors of individuals to more healthful ones. These limitations may be attributed, in part, to a lack of attention given to enhancing an individual’s psychosocial process variables, such as resilience, coping skills, self-leadership, and empowerment. Incorporating resilience education into lifestyle change therapies is a novel approach that addresses the behavior modification limitations of current interventions by aiming to enhance psychosocial process variables. Therefore, the purpose of this project was to conduct a six-month pilot study to determine the feasibility of our resilience and diabetes self-management intervention, The Diabetes Coaching Program: Transforming Lives Through Resilience Education, in a convenience sample of African American adults (n=16) with T2DM. The intervention included four weekly resilience and diabetes education classes and eight bi-weekly support group sessions. Survey data and blood samples were collected at baseline and at six months. Twelve participants completed the study (75% retention). Results indicated that higher perceived stress scores were associated with less resilience, fewer adaptive coping skills, lower self-leadership, lower diabetes empowerment and greater depressive symptoms. However, diabetes empowerment was the only psychosocial process variable to be significantly enhanced by the intervention at six months. Weight, BMI, HbA1c, total cholesterol, LDL cholesterol, blood pressure, and IGF-1 levels were significantly decreased at six months, whereas, lymphocyte proliferation and physical activity were significantly increased. These data |
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Cardiometabolic effects of a carbohydrate restricted diet and increased physical activity in men and women taking statins. $49.99 The effect of carbohydrate restriction and increased physical activity on statin treatment has not been examined. The objective of this study was to assess changes in LDL particle size, metabolic syndrome, and emerging cardiometabolic risk factors after a 6-week intervention. Twenty one men and postmenopausal women (mean+/-SD; age: 59.2+/-9.5 y, body weight: 89.2+/-16.8 kg, % body fat: 38.5+/-6.2%, body mass index: 29.52+/-3.03 kg/m 2) who had successfully lowered LDL-C with a statin (<130 mg/dl) were matched on sex, age, lipids, and type of statin and randomized to a very low carbohydrate ketogenic diet (LCD) or a low carbohydrate diet with increased physical activity (LCD+PA). Subjects received weekly dietary counseling from a registered dietitian and they were instructed to maintain body weight. Fasting blood and physiological measurements were taken at baseline, week 3, and week 6. LDL size was measured with non-gradient polyacrylamide gel electrophoresis and vertical auto profile ultracentrifugation. Food records (mean+/-SD; baseline: 2159+/-415 kcals, 45/36/20 %carb/fat/pro; intervention: 1739+/-481 kcals, 11/58/26 %carb/fat/pro), urine acetoacetic acid, and serum total ketones demonstrated dietary compliance. LCD+PA incrementally increased average daily steps using a pedometer, reaching 4,792 steps greater than baseline at week 6. LDL particle size (p≤0.05) and reactive hyperemia peak forearm blood flow significantly increased (p≤0.01) and TAG (-36%), insulin (-25%), systolic (-5%) and diastolic (-6%) blood pressure, body weight (-3%), and waist circumference (-4%) decreased. Change in body weight was not significantly correlated with changes in blood markers. There were no significant differences observed for total cholesterol, LDL-C, HDL-C, Lp(a), glucose, or flow-mediated dilation, nor were group differences found. These findings indicate that individuals on statin medication showing signs of metabolic syndrome respond favorably to a low |
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Cardiometabolic effects of a carbohydrate restricted diet and increased physical activity in men and women taking statins. $49.99 The effect of carbohydrate restriction and increased physical activity on statin treatment has not been examined. The objective of this study was to assess changes in LDL particle size, metabolic syndrome, and emerging cardiometabolic risk factors after a 6-week intervention. Twenty one men and postmenopausal women (mean+/-SD; age: 59.2+/-9.5 y, body weight: 89.2+/-16.8 kg, % body fat: 38.5+/-6.2%, body mass index: 29.52+/-3.03 kg/m 2) who had successfully lowered LDL-C with a statin (<130 mg/dl) were matched on sex, age, lipids, and type of statin and randomized to a very low carbohydrate ketogenic diet (LCD) or a low carbohydrate diet with increased physical activity (LCD+PA). Subjects received weekly dietary counseling from a registered dietitian and they were instructed to maintain body weight. Fasting blood and physiological measurements were taken at baseline, week 3, and week 6. LDL size was measured with non-gradient polyacrylamide gel electrophoresis and vertical auto profile ultracentrifugation. Food records (mean+/-SD; baseline: 2159+/-415 kcals, 45/36/20 %carb/fat/pro; intervention: 1739+/-481 kcals, 11/58/26 %carb/fat/pro), urine acetoacetic acid, and serum total ketones demonstrated dietary compliance. LCD+PA incrementally increased average daily steps using a pedometer, reaching 4,792 steps greater than baseline at week 6. LDL particle size (p≤0.05) and reactive hyperemia peak forearm blood flow significantly increased (p≤0.01) and TAG (-36%), insulin (-25%), systolic (-5%) and diastolic (-6%) blood pressure, body weight (-3%), and waist circumference (-4%) decreased. Change in body weight was not significantly correlated with changes in blood markers. There were no significant differences observed for total cholesterol, LDL-C, HDL-C, Lp(a), glucose, or flow-mediated dilation, nor were group differences found. These findings indicate that individuals on statin medication showing signs of metabolic syndrome respond favorably to a low |
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Cholesterol Revitaliser $13.95 About one in every three people in the US, UK and Europe have elevated cholesterol. So there is a very good chance that you have raised cholesterol and may not even know it.It is a silent killer, and you need to find out NOW how to combat it.”Cholesterol Revitaliser” is a fast-track health solution that can help you to do exactly that. It covers everything you need to know about effectively lowering your cholesterol.It explores the information that you need to make informed, intelligent decisions about your health.It discusses natural ways that you can dramatically lower your cholesterol, as well as the role that statins and supplements can play. It outlines diet choices to lower cholesterol naturally and explores the amazing variety of foods that can re-invigorate your system.Cholesterol Revitaliser covers everything that you need to know about effectively lowering your cholesterol, revitalising your health and cutting your chances of getting heart disease and heart attacks. Dramatically.Throughout Cholesterol Revitaliser you will find hard facts backed up by scientific studies. Facts. Not theory. In language that you can actually understand.By the end you will know these 7 things that you have probably never heard anywhere else before:1/ The Fat Lowdown… And How Swapping 30 Calories of One Dietary Substance for Another DOUBLES Your Risk of Heart Disease! (If you know what it is you can avoid it)2/ 110,000 People Can’t Be Wrong… The SHOCKING Statistic that could cut your risk of heart attack by 30%!3/ A ‘Food’ That Is 32% Fat, and Yet LOWERS YOUR CHOLESTEROL! (I Delve Into the Little Known Study That Tells All)4/ A Treatment That Lowers LDL Cholesterol by 8.7% in MONTH 1 and 15% by Month 4 and Makes HDL Cholesterol Rise by 10%! (And it doesn’t have the side effects of many commercial treatments!)5/ Another ‘Natural’ Treatment that could reduce your LDL Cholesterol by 10 to 20%. Reduce Triglyceride’s by 20 to 50% |
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Effect of carbohydrate restriction and American Heart Association diets on the clinical features of the metabolic syndrome, the inflammatory response and lipoprotein metabolism in young Emirati adults. $49.99 The incidence of diabetes mellitus (DM) in the UAE is one of the highest world-wide. The metabolic syndrome (MetS) characterized by central obesity, high blood pressure and dyslipidemias predisposes for the development of DM. The presence of MetS was assessed in a population of overweight/obese individuals by randomly screening 227 subjects, 18-50 (30.2 +/- 8.4 y), from Al-Ain city, Emirate of Abu Dhabi. 92 subjects (40.5%) were identified as having MetS. The most relevant clinical criteria associated with MetS were large waist circumference (WC), high systolic blood pressure and low HDL cholesterol (HDL-C).;Dietary interventions were conducted in 39 subjects. Initially, all individuals followed a carbohydrate restricted diet (CRD) with 25% energy from carbohydrate. After 6 wk, half of the subjects (n=19) were switched to the low fat diet (55% energy from carbohydrate) recommended by the American Heart Association (AHA group) while the other half (n=20) continued with the CRD diet for additional 6 wk (CRD group). Diet records indicated high compliance with the dietary guidelines. At wk 6, all subjects presented decreases in body weight (P < 0.0001), WC (P < 0.001), body fat (P < 0.0001) and plasma triglycerides (TG) (P < 0.0001). Significant decreases were also seen in plasma LDL cholesterol, blood pressure, insulin and inflammation markers while a significant increase in adiponectin was observed. After 12 wk, these positive changes persisted for all subjects independent of diet. However, body weight, plasma TG, insulin and plasma glucose were lower in the CRD (P < 0.05) compared to the AHA group.;The large atherogenic VLDL subfraction was decreased over time for all subjects (P < 0.01) with a more pronounced decrease (P <0.05) in the CRD compared to the AHA group. Medium and small LDL particles and apolipoprotein B decreased for all subjects (P < 0.01) rendering a less atherogenic lipoprotein profile. These studies indicate that CRD can |
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Effect of carbohydrate restriction and American Heart Association diets on the clinical features of the metabolic syndrome, the inflammatory response and lipoprotein metabolism in young Emirati adults. $49.99 The incidence of diabetes mellitus (DM) in the UAE is one of the highest world-wide. The metabolic syndrome (MetS) characterized by central obesity, high blood pressure and dyslipidemias predisposes for the development of DM. The presence of MetS was assessed in a population of overweight/obese individuals by randomly screening 227 subjects, 18-50 (30.2 +/- 8.4 y), from Al-Ain city, Emirate of Abu Dhabi. 92 subjects (40.5%) were identified as having MetS. The most relevant clinical criteria associated with MetS were large waist circumference (WC), high systolic blood pressure and low HDL cholesterol (HDL-C).;Dietary interventions were conducted in 39 subjects. Initially, all individuals followed a carbohydrate restricted diet (CRD) with 25% energy from carbohydrate. After 6 wk, half of the subjects (n=19) were switched to the low fat diet (55% energy from carbohydrate) recommended by the American Heart Association (AHA group) while the other half (n=20) continued with the CRD diet for additional 6 wk (CRD group). Diet records indicated high compliance with the dietary guidelines. At wk 6, all subjects presented decreases in body weight (P < 0.0001), WC (P < 0.001), body fat (P < 0.0001) and plasma triglycerides (TG) (P < 0.0001). Significant decreases were also seen in plasma LDL cholesterol, blood pressure, insulin and inflammation markers while a significant increase in adiponectin was observed. After 12 wk, these positive changes persisted for all subjects independent of diet. However, body weight, plasma TG, insulin and plasma glucose were lower in the CRD (P < 0.05) compared to the AHA group.;The large atherogenic VLDL subfraction was decreased over time for all subjects (P < 0.01) with a more pronounced decrease (P <0.05) in the CRD compared to the AHA group. Medium and small LDL particles and apolipoprotein B decreased for all subjects (P < 0.01) rendering a less atherogenic lipoprotein profile. These studies indicate that CRD can |
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Effect of varying the fatty acid composition of a carbohydrate-restricted diet on plasma fatty acid composition, blood lipids oxidative stress, inflammation and insulin sensitivity in men. $49.99 Background. Carbohydrate-restricted diets (CRD) consistently improve risk factors associated with Metabolic Syndrome (MetSyn). Recently, we showed that a saturated-fat rich hypocaloric CRD significantly reduced serum content of saturated fatty acids (SFA) and significantly increased arachidonic acid (AA) and the omega-6/omega-3 polyunsaturated (PUFA) ratio while significantly decreasing inflammation compared to a low-fat diet. This disconnect between dietary and circulating lipid lead us to explore how varying the fat composition of a CRD affects these variables, in addition to oxidative stress. Methods. Eight healthy weight-stable men (age 45 +/- 7.9 y, body fat 28.4 +/- 6.5%) were fed two eucaloric CRD varying in saturated fat and unsaturated fat with the same macronutrient distribution (12%en carbohydrate, 30%en protein, 58%en fat, 850 mg cholesterol) for 6 weeks each without weight loss. Similar foods were fed but CRD-SFA emphasized dairy fat while CRD-UFA emphasized olive oil, omega-3 fortified eggs, salmon, and walnuts. CRD-SFA provided almost twice as much SFA (86 g vs 47 g) and less monounsaturated fat (MUFA) and omega-6 and omega-3 PUFA than CRD-UFA, confirmed by chemical analysis. Fasting blood and 24 hr urine was analyzed at baseline and following each diet for fasting lipoproteins, insulin, glucose, inflammatory markers, fatty acid composition in plasma triacylglycerides (TAG), phospholipids (PL) and cholesteryl esters (CE), and urine 8-iso PGF2alpha. Results. Regardless of fat quality, both CRD significantly decreased TAG and insulin, and increased HDL-C and LDL particle size (P < 0.05). Despite increased total-cholesterol (TC) and LDL-cholesterol (LDL-C) after CRD-SFA, the TC/LDL-C ratio was not different between diets. Total plasma SFA in CE, PL, and TAG were unchanged after the CRD-SFA, and plasma PL and CE AA content was significantly increased. CRD-UFA significantly increased PL and CE EPA+DHA content and the PL omega-3 index. Inflammation was |
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Effect of varying the fatty acid composition of a carbohydrate-restricted diet on plasma fatty acid composition, blood lipids oxidative stress, inflammation and insulin sensitivity in men. $49.99 Background. Carbohydrate-restricted diets (CRD) consistently improve risk factors associated with Metabolic Syndrome (MetSyn). Recently, we showed that a saturated-fat rich hypocaloric CRD significantly reduced serum content of saturated fatty acids (SFA) and significantly increased arachidonic acid (AA) and the omega-6/omega-3 polyunsaturated (PUFA) ratio while significantly decreasing inflammation compared to a low-fat diet. This disconnect between dietary and circulating lipid lead us to explore how varying the fat composition of a CRD affects these variables, in addition to oxidative stress. Methods. Eight healthy weight-stable men (age 45 +/- 7.9 y, body fat 28.4 +/- 6.5%) were fed two eucaloric CRD varying in saturated fat and unsaturated fat with the same macronutrient distribution (12%en carbohydrate, 30%en protein, 58%en fat, 850 mg cholesterol) for 6 weeks each without weight loss. Similar foods were fed but CRD-SFA emphasized dairy fat while CRD-UFA emphasized olive oil, omega-3 fortified eggs, salmon, and walnuts. CRD-SFA provided almost twice as much SFA (86 g vs 47 g) and less monounsaturated fat (MUFA) and omega-6 and omega-3 PUFA than CRD-UFA, confirmed by chemical analysis. Fasting blood and 24 hr urine was analyzed at baseline and following each diet for fasting lipoproteins, insulin, glucose, inflammatory markers, fatty acid composition in plasma triacylglycerides (TAG), phospholipids (PL) and cholesteryl esters (CE), and urine 8-iso PGF2alpha. Results. Regardless of fat quality, both CRD significantly decreased TAG and insulin, and increased HDL-C and LDL particle size (P < 0.05). Despite increased total-cholesterol (TC) and LDL-cholesterol (LDL-C) after CRD-SFA, the TC/LDL-C ratio was not different between diets. Total plasma SFA in CE, PL, and TAG were unchanged after the CRD-SFA, and plasma PL and CE AA content was significantly increased. CRD-UFA significantly increased PL and CE EPA+DHA content and the PL omega-3 index. Inflammation was |
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Effectiveness of a web-based nutrition education program to reduce cardiovascular disease risk among U.S. army personnel and their families (”Defend Your Heart” study). $49.99 Cardiovascular disease (CVD) continues to be the leading cause of mortality and morbidity in the United States. Military personnel are also vulnerable to this killer disease due to indulgence in unhealthy behaviors such as cigarette smoking, physical inactivity, poor nutrition, and high stress. Formative assessment identified a need for web-based resources for the Army registered dietitians (RDs) and for deployed or remotely located military beneficiaries. The purpose of the current study was to create and assess the effectiveness of a web-site “Defend Your Heart”. This web-site was targeted to two audiences: RDs and a self care program for the military beneficiaries. This self care program was created using the framework of Rosenstock’s Expanded Health Belief Model (EHBM).;The effectiveness of a web-based self care program was evaluated using a randomized 4-month study with participants either in the web-based group (n=17) or the usual care (n=13) at a U.S. Army hospital. Data were collected at baseline, two months and four months. Variables measured were anthropometric, blood pressure, lipid profile, fasting glucose, C-reactive protein, nutrient intake, physical activity, and EHBM constructs. Data were analyzed using analysis of covariance and using baseline means to adjust the two and four month data. Results indicated a significant reduction of total blood cholesterol, LDL-cholesterol, predicted body fat percent, and estimated body mass index (P<0.05) in the web-based group. The usual group demonstrated a significant increase in self-efficacy score at month four (p<0.05).;Significant within group changes for both groups were demonstrated for the reduction in waist circumference and serum triglycerides (p<0.05). Due to a smaller sample size caution is required while interpreting the results. The results of the web-site usability showed that a majority of the RDs (n=34) and web-based participants (n=8) were satisfied with the content and ease of |
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Effectiveness of a web-based nutrition education program to reduce cardiovascular disease risk among U.S. army personnel and their families (”Defend Your Heart” study). $49.99 Cardiovascular disease (CVD) continues to be the leading cause of mortality and morbidity in the United States. Military personnel are also vulnerable to this killer disease due to indulgence in unhealthy behaviors such as cigarette smoking, physical inactivity, poor nutrition, and high stress. Formative assessment identified a need for web-based resources for the Army registered dietitians (RDs) and for deployed or remotely located military beneficiaries. The purpose of the current study was to create and assess the effectiveness of a web-site “Defend Your Heart”. This web-site was targeted to two audiences: RDs and a self care program for the military beneficiaries. This self care program was created using the framework of Rosenstock’s Expanded Health Belief Model (EHBM).;The effectiveness of a web-based self care program was evaluated using a randomized 4-month study with participants either in the web-based group (n=17) or the usual care (n=13) at a U.S. Army hospital. Data were collected at baseline, two months and four months. Variables measured were anthropometric, blood pressure, lipid profile, fasting glucose, C-reactive protein, nutrient intake, physical activity, and EHBM constructs. Data were analyzed using analysis of covariance and using baseline means to adjust the two and four month data. Results indicated a significant reduction of total blood cholesterol, LDL-cholesterol, predicted body fat percent, and estimated body mass index (P<0.05) in the web-based group. The usual group demonstrated a significant increase in self-efficacy score at month four (p<0.05).;Significant within group changes for both groups were demonstrated for the reduction in waist circumference and serum triglycerides (p<0.05). Due to a smaller sample size caution is required while interpreting the results. The results of the web-site usability showed that a majority of the RDs (n=34) and web-based participants (n=8) were satisfied with the content and ease of |
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Familial Hypercholesterolemia $53.23 Familial hypercholesterolemia (abbreviated FH, also spelled familial hypercholesterolaemia) is a genetic disorder characterized by high cholesterol levels, specifically very high low-density lipoprotein (LDL, “bad cholesterol”) levels, in the blood and early cardiovascular disease. Many patients have mutations in the LDLR gene that encodes the LDL receptor protein, which normally removes LDL from the circulation, or apolipoprotein B (ApoB), which is the part of LDL that binds with the receptor; mutations in other genes are rare. Patients who have one abnormal copy (are heterozygous) of the LDLR gene may have premature cardiovascular disease at the age of 30 to 40. Having two abnormal copies (being homozygous) may cause severe cardiovascular disease in childhood. Heterozygous FH is a common genetic disorder, occurring in 1:500 people in most countries; homozygous FH is much rarer, occurring in 1 in a million births. Heterozygous FH is normally treated with statins, bile acid sequestrants or other hypolipidemic agents that lower cholesterol levels. |
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Fenofibrate $43.04 High Quality Content by WIKIPEDIA articles! Fenofibrate is a drug of the fibrate class. Fenofibrate was discovered by Groupe Fournier SA, before it was acquired in 2005 by Solvay Pharmaceutical, a business unit owned by the Belgian corporation, Solvay S.A. It is mainly used to reduce cholesterol levels in patients at risk of cardiovascular disease. Like other fibrates, it reduces both low-density lipoprotein (LDL) and very low density lipoprotein (VLDL) levels, as well as increasing high-density lipoprotein (HDL) levels and reducing tryglycerides level. |
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Guggul: Ayurveda’s Wonder Herb $1.99 Guggul (Commiphora mukul), long used by ayurvedic practitioners, possesses strong rejuvenating and purifying qualities. A potent antioxidant, guggul acts to lower “bad” LDL cholesterol levels and raise “good” HDL cholesterol levels. In addition, guggul has strong anti-inflammatory properties that can be useful in treating arthritis and other chronic inflammatory conditions. |
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How to Get in Shape Fast! $4.99 Want to learn how to get in shape fast? Looking for the best way to get in shape? Desirous to get in shape for summer? Maybe it’s time you picked up a copy of How to Get in the Shape Fast! and start learning ways to get in shape and get in shape fast.If you have no time in your day . . . If you don’t know the difference between a barbell and a cowbell . . . You can find a way to add simple, quick exercises to your daily regimens for a slimmer waistline, increased energy, and a happier life!It can be hard to get to the gym after a long day at work. Your family expects you back as soon as possible. You’re pent up in a cubicle all day, never able to stretch. Or constantly leaving for business trips.Anyone who’s ever been in your shoes knows it can be difficult to keep a regular workout routine.The truth is, you can get exercise without working out. You can now get in shape fast!You’re about to discover how you can fit simple, quick exercises into your day and get in shape quick…even if you have to multi-task.You can melt away those extra pounds, increase your energy and feel good about your health.Learn all the tips, tricks, and tactics of how to get in shape quick from those who have to be creative to stay active. You’re about to discover :*A new way to look at how you treat and care for your body*5 ways people end up looking older than they really are and how you can avoid it*3 questions you must ask yourself to honestly judge how fit you are*The secret behind “slowly but surely” and how it can save the day for people on the go*5 reasons you’ll never regret getting in shape*The lowdown on HDL/LDL counts, your cholesterol, and the risks of being in the red zone*If you’re a woman, exercise can be one of your greatest weapons against osteoporosis*How to prevent diabetes with exercise*How being |
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Qadir’s Test $49.99 Please note that the content of this book primarily consists of articles available from Wikipedia or other free sources online. High Quality Content by WIKIPEDIA articles! Qadir’s test is used for the diagnosis of cancer. The plasma lipid profile is changed in cancer patients. So the change in plasma lipid profile may be used as marker for diagnosis of cancer. The test was named by a scientist Dr. M.I. Qadir who worked on it. Lipids are carried in body fluids with the help of lipoproteins, chylomicrons transport of triglycerides from the intestine to all cells. Very low-density lipoproteins (VLDL) are involved in the transportation of triglycerides from the liver to other cells. Low-density lipoproteins (LDL) are responsible for the transport of cholesterol from liver to the cells and high density lipoproteins (HDL) are involved for the transport of cholesterol from cells to the liver. Chylomicrons and very low density lipoproteins are rapidly catabolized. Thus triglycerides, cholesterol, LDL-cholesterol and HDL-cholesterol constitute Plasma Lipid Profile. |
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The relationship between calcium intake, obesity, and cardiovascular disease risk factors: The Jackson Heart Study. $49.99 Cardiovascular disease (CVD) is a major health risk in the United States. Major indicators of CVD risk include obesity, blood lipids, and blood pressure. Modifiable risk factors associated with CVD include body composition (body mass index and waist circumference), serum lipids, and blood pressure. Data suggest calcium intake may play a role in regulation of weight, serum lipids, and blood pressure. The purpose of this study was to assess relationships of dietary calcium intake with weight status, and cardiovascular disease risks in African American population participating in the Jackson Heart Study. The subjects included 4,267 African American adults ages 21-95 years (mean = 55.1+/-12.6 years) in the Jackson Heart Study (JHS). Dependent variables included: body mass index (BMI) calculated from measured height/weight (stadiometer/balance scale), waist circumference (WC; measuring tape), serum lipids, and blood pressure (sphygmomanometer). A 158-item food frequency questionnaire (FFQ) was used to assess nutrient intake. Statistical analyses included multiple regression analysis and Pearson correlations using SPSS 16.0 (SPSS Inc. Chicago, IL, USA). There was a significant positive relationship between calcium intake and the body composition measure BMI [F (4, 3982) = 3.26, p = 0.019, DeltaR2 = .003] and a significant inverse relationship between calcium intake and WC [F (4, 3982) = 2.43, p = 0.05, DeltaR 2 = .002]. These relationships were also observed in females only when data were analyzed by gender. There were significant inverse relationships between calcium intake and total cholesterol (TC) [F (4, 4259 = 5.46, p = 0.266, DeltaR2 = .002] and LDL-cholesterol (LDL) [F (4, 4225) = 3.218 p = 0.01, DeltaR2 = .003]. There were also significant inverse correlations between total cholesterol (TC) and LDL and calcium for males only. There was a significant relationship between calcium intake and HDL-cholesterol [F (4, 4259) = 13.31, p < 0.001, DeltaR2 = .012], as |
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The relationship between calcium intake, obesity, and cardiovascular disease risk factors: The Jackson Heart Study. $49.99 Cardiovascular disease (CVD) is a major health risk in the United States. Major indicators of CVD risk include obesity, blood lipids, and blood pressure. Modifiable risk factors associated with CVD include body composition (body mass index and waist circumference), serum lipids, and blood pressure. Data suggest calcium intake may play a role in regulation of weight, serum lipids, and blood pressure. The purpose of this study was to assess relationships of dietary calcium intake with weight status, and cardiovascular disease risks in African American population participating in the Jackson Heart Study. The subjects included 4,267 African American adults ages 21-95 years (mean = 55.1+/-12.6 years) in the Jackson Heart Study (JHS). Dependent variables included: body mass index (BMI) calculated from measured height/weight (stadiometer/balance scale), waist circumference (WC; measuring tape), serum lipids, and blood pressure (sphygmomanometer). A 158-item food frequency questionnaire (FFQ) was used to assess nutrient intake. Statistical analyses included multiple regression analysis and Pearson correlations using SPSS 16.0 (SPSS Inc. Chicago, IL, USA). There was a significant positive relationship between calcium intake and the body composition measure BMI [F (4, 3982) = 3.26, p = 0.019, DeltaR2 = .003] and a significant inverse relationship between calcium intake and WC [F (4, 3982) = 2.43, p = 0.05, DeltaR 2 = .002]. These relationships were also observed in females only when data were analyzed by gender. There were significant inverse relationships between calcium intake and total cholesterol (TC) [F (4, 4259 = 5.46, p = 0.266, DeltaR2 = .002] and LDL-cholesterol (LDL) [F (4, 4225) = 3.218 p = 0.01, DeltaR2 = .003]. There were also significant inverse correlations between total cholesterol (TC) and LDL and calcium for males only. There was a significant relationship between calcium intake and HDL-cholesterol [F (4, 4259) = 13.31, p < 0.001, DeltaR2 = .012], as |
Category: Cholesterol Diet
