Hdl Cholesterol
How to Raise HDL Cholesterol Levels
There are two types of Cholesterol Levels known as low-density lipoprotein (LDL), and high-density lipoprotein (HDL). HDL is the Good Cholesterol, and LDL is the bad, which can be lowered naturally by raising the good cholesterol levels. As the LDL makes its way into the body, the HDL helps to round it up and eliminate it from the body. Therefore it is essential to raise HDL cholesterol levels in order to prevent high Ldl Cholesterol.
Healthy Fats
Monounsaturated fats have been shown to raise the good cholesterol levels naturally when added to a daily diet. These fats can be obtained in abundance from avocados and nuts and seeds, as well as in smaller amounts from leafy greens. A leafy green salad topped with avocados and nuts and seeds is a good way to balance cholesterol levels.
Saturated fats should be avoided or at least be reduced, as they are commonly associated with low HDL levels. These are fats that come from meats, particularly red meat, dairy and eggs, as well as many processed foods.
Consume Fiber
Foods high in fiber, such as fruits and vegetables and nuts and seeds also help to raise HDL cholesterol levels. The fiber locks onto the Bad Cholesterol in the digestive tract and eliminates it from the body before it can be absorbed. Thus the good cholesterol levels can be raised.
Consume Niacin Rich Foods
Niacin is a popular drug that is used to treat High Cholesterol Levels. Niacin is also known as vitamin B3, and it can be found in many plant-based foods such as cashews, flax seeds, pecans, almonds, sunflower seeds, mushrooms, beans and leafy greens. Studies show that by adding these foods to the diet, they can help to raise the HDL cholesterol.
Exercise
Regular exercise has also been shown to help raise the HDL levels by helping the body to sweat out harmful toxins. Exercise in turn can help to lose weight, which is also essential in raising the good cholesterol levels.
Avoid Stimulants
Stimulants such as alcohol, cigarettes and caffeine have been shown to interfere negatively with cholesterol levels.
Reduce Stress
Stress can also interfere with cholesterol levels, as it often causes individuals to reach for comfort foods such as chocolate, ice cream, cake or other unhealthy foods. These types of comfort foods contribute to high levels of triglycerides, which in turn can cause the HDL levels to drop.
When cholesterol levels are out of balance it can lead to many artery problems, heart disease and stroke.
About the Author
To find out more about raw and living foods visit http://www.livingfood101.com/. You can also join me at http://hubpages.com/profile/LivingFood and http://www.triond.com/users/LivingFood
Dysfunctional HDL Cholesterol Risks
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Dietary Management of Fat and Cholesterol (Video Counseling Library, Cardiology) This video explains how HDL, LDL, saturated fats, andunsaturated fats affect the body, and the importance ofreducing cholesterol levels. Viewers learn American Heart Association based diet strategies for healthier foods, howto read labels, and how to prepare foods…. |
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Heart Decisions For Life: Compliance (Cholesterol Watch) Discusses why compliance is so critical to regulating cholesterol. You must be an active participant in your own health to succeed in controlling your cholesterol…. |
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Cholesterol: Raising the Good and Lowering the Bad $9.99 … |
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Managing Cholesterol (Home Use) $24.95 Part of the award winning public television series Healthy Body/Healthy Mind. Heart disease is the number one killer of men and women in America and high cholesterol numbers are a major risk factor for both heart attacks and strokes. In this program we offer information about lifestyle changes that can help keep cholesterol numbers in the normal range, plus we see how changing the diet and adding… |
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Cholesterol: Raising the Good and Lowering the Bad (Home Use) $24.95 By now, most people have heard the term HDL – good cholesterol, and LDL – bad cholesterol. But do you know what makes good cholesterol good and bad cholesterol bad? And how does the wrong combination of these lipoproteins affect the risk of heart disease? This program will delve into these important questions and provide you with the answers you need to control your cholesterol.This product is man… |
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Himalaya Shuddha Guggulu Cholesterol Regulator 60 Capsules $0.01 The Archer Farms brand is known for its superior-quality, unique product offerings including a wide variety of gourmet groceries, appetizers and European-style baked goods…. |
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100% Japanese Sencha Blended With Matcha Green Tea Powder – 5.3 Oz Loose Tea 100% Japanese Green tea blended with Matcha green tea powder for its wonderful taste and beautiful presentation…. |
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All Natural Cold Pressed Organic Perilla Oil – Unfiltered, Unrefined – 250 Ml (8.3 Oz) Perilla oil, extracted from the plant fructescens, is a rich source of alpha-linolenic acid (ALA). ALA is precursor to the omega-3 fatty acids EPA and DHA. ALA is similar to fish oil in its benefits, but without any unpleasant aftertaste. ALA may support the health if many organs and body systems, including the cardiovascular system, immune system, skin, brain, and joints. Population and animal st… |
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BMV Quantum Subliminal CD Lower Cholesterol: Low Cholesterol Mind Program (Ultrasonic Subliminal Series) $14.99 Program your subconscious mind to lower cholesterol levels. Create life-changing results using state-of-the-art subliminal and brainwave entrainment technologies. Tune your brainwaves to specific frequencies by listening to this CD! Program your subconscious mind for positive lasting results, created by a Certified Hypnotherapist and NLP Practitioner (Neuro-Linguistic Programming). Silent affirmat… |
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CholestOff … |
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101 Ways to Lower Your Cholesterol: Easy Tips that Allow You to Take Control, Reduce Risk, and Live Longer $9.95 Today, more than ever, people are thinking about their cholesterol levels. But the resources they’ve had to help them be healthy were intimidating and expensive–until now.This guide is the non-intimidating, easy-to-follow, one-stop resource for managing and maintaining healthy HDL, or “good” cholesterol levels.From recognizing the risks of high cholesterol and artery blockage to creating a sound diet and exercise plan, this is a comprehensive yet uncomplicated guide. In bite-sized tips, it provides the keys to lowering the risk of heart disease–the leading killer of men and women in the United States.Helping you take the steps you need to live a healthier lifestyle, this tip-based book is the ultimate collection of life-saving suggestions for anyone affected by bad cholesterol. |
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101 Ways to Lower Your Cholesterol: Easy Tips that Allow You to Take Control, Reduce Risk, and Live Longer $1.63 Today, more than ever, people are thinking about their cholesterol levels. But the resources they’ve had to help them be healthy were intimidating and expensive–until now.This guide is the non-intimidating, easy-to-follow, one-stop resource for managing and maintaining healthy HDL, or “good” cholesterol levels.From recognizing the risks of high cholesterol and artery blockage to creating a sound diet and exercise plan, this is a comprehensive yet uncomplicated guide. In bite-sized tips, it provides the keys to lowering the risk of heart disease–the leading killer of men and women in the United States.Helping you take the steps you need to live a healthier lifestyle, this tip-based book is the ultimate collection of life-saving suggestions for anyone affected by bad cholesterol. |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $49.99 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $49.99 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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A comprehensive dose-response study of the effects of pistachios on cardiovascular disease risk factors: A translational research approach integrating clinical nutrition and molecular biology. $108 Nut consumption reduces risk for cardiovascular disease (CVD). Few studies have evaluated the effects of pistachios on CVD risk factors and they have not evaluated dose-response relationships or lipid-lowering mechanisms. Nutrition studies with a translational research approach integrate clinical nutrition and molecular biology, allowing for the investigation of clinical responses and underlying cellular mechanisms. The present study utilized a translational research approach to comprehensively evaluate the effects of pistachios on CVD. We employed a randomized crossover controlled-feeding study to evaluate the effects of two doses of pistachios, added to a lower-fat diet, on lipids and lipoproteins, apolipoprotein-defined lipoprotein subclasses, and plasma fatty acids. To investigate mechanisms of action, we measured serum cholesteryl ester transfer protein (CETP), indices of plasma stearoyl-CoA desaturase activity (SCD), and gene expression in isolated peripheral blood mononuclear cells (PBMCs). Total cholesterol (TC), LDL-C, non-HDL-C, apoB, and apoB/apoA-I decreased after both pistachio diets; and triacylglycerol and plasma SCD activity decreased after the 3.0 ounce pistachio diet (P < 0.05). Pistachios elicited a dose-dependent lowering of TC/HDL-C, LDL-C/HDL-C, and non-HDL-C/HDL-C (P < 0.01). We evaluated the effects of pistachios on expression of genes related to inflammation and lipid metabolism (TNFalpha, IL-1beta, IL-6, ICAM, VCAM, CETP, and LCAT) in PBMCs. Furthermore, we investigated the relationship between diet-induced change in CETP expression and change in serum CETP and plasma lipids/lipoproteins. The pistachio-rich diets significantly decreased IL-1beta expression compared to baseline (P < 0.05). Change in CETP expression in PBMCs predicted change in LDL-C, NONHDL-C, TC/HDL-C, and NONHDL-C/HDL-C in individuals who were diet-responsive with regards to serum CETP. In conclusion, this study demonstrates that pistachios elicit beneficial |
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Apoa-1 Milano $44.13 High Quality Content by WIKIPEDIA articles! ApoA-1 Milano is a naturally occurring mutated variant of the apolipoprotein A1 protein found in human HDL, the lipoprotein particle that carries cholesterol from tissues to the liver and is associated with protection against cardiovascular disease. ApoA1 Milano was first identified by Dr Cesare Sirtori in Milan, who also demonstrated that its presence significantly reduced cardiovascular disease, even though it caused a reduction in HDL levels and an increase in triglyceride levels. |
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Application of the Mediterranean-style diet principles to the American diet: Does a diet consistent with the Mediterranean-style diet protect against the development of risk factors for type 2 diabetes mellitus in the Framingham Offspring Cohort? $49.99 The objective of the project was to examine the relationship between a diet consistent with Mediterranean-style dietary pattern and the metabolic risk factors of type 2 diabetes mellitus (DM), a major risk factor for cardiovascular disease (CVD). The objective of this project was accomplished using data from the Framingham Heart Study Offspring Cohort.;A Mediterranean-Style Dietary Pattern Score (MSDPS) was created to assess the conformity of an individual’s diet to a traditional Mediterranean-style diet. This continuously-scaled score (range 0–100), was based on adherence to recommended intakes levels from the Mediterranean diet pyramid and took into account over-consumption of foods and consumption of foods that are not on the Mediterranean pyramid. In 3030 participants, the MSDPS demonstrated content validity against nutrients known to be associated with the Mediterranean-style dietary pattern, including expected positive associations with dietary fiber, n-3 fatty acids, antioxidant vitamins, calcium, magnesium and potassium, and inverse associations with added sugar, glycemic index, saturated fat, trans-fat and n-6:n-3 fatty acid ratio.;The longitudinal association over 7 years (mean follow-up = 7 yr) between MSDPS and insulin resistant traits (HOMA-IR, fasting glucose, triglyceride, HDL-cholesterol, blood pressure) and incidence of metabolic syndrome was examined in 2,388 participants without DM and 1,820 participants free of DM and metabolic syndrome at baseline, respectively. The MSDPS was inversely associated with HOMA-IR (P-trend=0.02), waist circumference (P-trend <0.001) and plasma triglyceride (P-trend<0.001); and was positively associated with HDL-cholesterol (P-trend=0.05). A higher MSDPS was also associated with an 8% lower incidence of metabolic syndrome (P-trend=0.03).;In participants without DM or clinically evident CVD, the cross-sectional association was examined between the MSDPS and endothelial function, as measured by brachial artery |
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Application of the Mediterranean-style diet principles to the American diet: Does a diet consistent with the Mediterranean-style diet protect against the development of risk factors for type 2 diabetes mellitus in the Framingham Offspring Cohort? $49.99 The objective of the project was to examine the relationship between a diet consistent with Mediterranean-style dietary pattern and the metabolic risk factors of type 2 diabetes mellitus (DM), a major risk factor for cardiovascular disease (CVD). The objective of this project was accomplished using data from the Framingham Heart Study Offspring Cohort.;A Mediterranean-Style Dietary Pattern Score (MSDPS) was created to assess the conformity of an individual’s diet to a traditional Mediterranean-style diet. This continuously-scaled score (range 0–100), was based on adherence to recommended intakes levels from the Mediterranean diet pyramid and took into account over-consumption of foods and consumption of foods that are not on the Mediterranean pyramid. In 3030 participants, the MSDPS demonstrated content validity against nutrients known to be associated with the Mediterranean-style dietary pattern, including expected positive associations with dietary fiber, n-3 fatty acids, antioxidant vitamins, calcium, magnesium and potassium, and inverse associations with added sugar, glycemic index, saturated fat, trans-fat and n-6:n-3 fatty acid ratio.;The longitudinal association over 7 years (mean follow-up = 7 yr) between MSDPS and insulin resistant traits (HOMA-IR, fasting glucose, triglyceride, HDL-cholesterol, blood pressure) and incidence of metabolic syndrome was examined in 2,388 participants without DM and 1,820 participants free of DM and metabolic syndrome at baseline, respectively. The MSDPS was inversely associated with HOMA-IR (P-trend=0.02), waist circumference (P-trend <0.001) and plasma triglyceride (P-trend<0.001); and was positively associated with HDL-cholesterol (P-trend=0.05). A higher MSDPS was also associated with an 8% lower incidence of metabolic syndrome (P-trend=0.03).;In participants without DM or clinically evident CVD, the cross-sectional association was examined between the MSDPS and endothelial function, as measured by brachial artery |
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Association Of Hypertension Diagnosis And Smoking Cessation $51.56 Hypertension and smoking are two major issues threatening the nation’s health. Previous studies examining their relationship have resulted in conflicting reports that leaves readers with the question: Does hypertension diagnosis induce one to quit smoking or does smoking cessation lead to behaviors that increase the risk of hypertension diagnosis? The aim of this work is to closely examine this relationship. Data from the Third National Health and Nutrition Examination Survey (NHANES III) were used in this investigation. Physical examination measurements of blood pressure and self-reported diagnosis and smoking behavior were used to define hypertension and smoking status. The risk of prior hypertension diagnosis associated with smoking cessation was estimated from a multivariate logistic regression model, adjusting for gender, age, ethnicity, BMI, physical activity, HDL cholesterol, and alcohol use. After adjusting for these confounders, are ex-smokers significantly more likely to have a prior hypertension diagnosis? Researchers in the fields of Public Health and Biostatistics will find the results astounding. |
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Asymptomatic Atherosclerosis: Pathophysiology, Detection and Treatment $239 Despite recent advances in the diagnosis and treatment of symptomatic atherosclerosis, available traditional screening methods for early detection and treatment of asymptomatic coronary artery disease are grossly insufficient and fail to identify the majority of victims prior to the onset of a life-threatening event. In Asymptomatic Atherosclerosis: Pathophysiology, Detection and Treatment, Dr. Morteza Naghavi and leading authorities from the Society for Heart Attack Prevention and Eradication (SHAPE) present a new paradigm for the screening and primary prevention of asymptomatic atherosclerosis. The text focuses on accurate, yet underutilized, measures of subclinical atherosclerosis, notably coronary artery calcium scanning and carotid intima-media thickness measurement. The authors introduce a comprehensive approach to identifying the vulnerable patients (asymptomatic individuals at risk of a near future adverse event). Additional chapters discuss future directions towards containing the epidemic of atherosclerotic cardiovascular disease using innovative solutions such as preemptive interventional therapies (bioabsorbable stents) for stabilization of vulnerable atherosclerotic plaques, mass unconditional Polypill therapy for population-based risk reduction, and ultimately vaccination strategies to prevent the development of atherosclerosis.Up-to-date and authoritative, Asymptomatic Atherosclerosis: Pathophysiology, Detection, and Treatment is a must-have for any cardiologist or primary care physician who wishes to practice modern preventive cardiology and manage the increasing number of asymptomatic atherosclerotic patients.• Outlines more accurate measures of risk (coronary artery calcium and carotid intima-media thickness) than traditional risk factors (total cholesterol, LDL cholesterol, HDL cholesterol)• Presents new multipronged strategies to aid in the early detection and treatment of high risk asymptomatic patients |
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Atkins Diabetes Revolution: The Groundbreaking Approach to Preventing and Controlling Type 2 Diabetes $12.99 Atkins Diabetes Revolution will help you:Find out if you have the metabolic syndromeIdentify your risk for diabetes as soon as possibleMake permanent lifestyle changes to normalize the underlying metabolic imbalances that lead to Type 2 diabetesReduce your chances of suffering a heart attack, stroke, and even some cancersReduce your risk of heart disease by lowering your triglycerides while increasing your HDL (“good”) cholesterol |
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Cardiometabolic effects of a carbohydrate restricted diet and increased physical activity in men and women taking statins. $49.99 The effect of carbohydrate restriction and increased physical activity on statin treatment has not been examined. The objective of this study was to assess changes in LDL particle size, metabolic syndrome, and emerging cardiometabolic risk factors after a 6-week intervention. Twenty one men and postmenopausal women (mean+/-SD; age: 59.2+/-9.5 y, body weight: 89.2+/-16.8 kg, % body fat: 38.5+/-6.2%, body mass index: 29.52+/-3.03 kg/m 2) who had successfully lowered LDL-C with a statin (<130 mg/dl) were matched on sex, age, lipids, and type of statin and randomized to a very low carbohydrate ketogenic diet (LCD) or a low carbohydrate diet with increased physical activity (LCD+PA). Subjects received weekly dietary counseling from a registered dietitian and they were instructed to maintain body weight. Fasting blood and physiological measurements were taken at baseline, week 3, and week 6. LDL size was measured with non-gradient polyacrylamide gel electrophoresis and vertical auto profile ultracentrifugation. Food records (mean+/-SD; baseline: 2159+/-415 kcals, 45/36/20 %carb/fat/pro; intervention: 1739+/-481 kcals, 11/58/26 %carb/fat/pro), urine acetoacetic acid, and serum total ketones demonstrated dietary compliance. LCD+PA incrementally increased average daily steps using a pedometer, reaching 4,792 steps greater than baseline at week 6. LDL particle size (p≤0.05) and reactive hyperemia peak forearm blood flow significantly increased (p≤0.01) and TAG (-36%), insulin (-25%), systolic (-5%) and diastolic (-6%) blood pressure, body weight (-3%), and waist circumference (-4%) decreased. Change in body weight was not significantly correlated with changes in blood markers. There were no significant differences observed for total cholesterol, LDL-C, HDL-C, Lp(a), glucose, or flow-mediated dilation, nor were group differences found. These findings indicate that individuals on statin medication showing signs of metabolic syndrome respond favorably to a low |
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Cardiometabolic effects of a carbohydrate restricted diet and increased physical activity in men and women taking statins. $49.99 The effect of carbohydrate restriction and increased physical activity on statin treatment has not been examined. The objective of this study was to assess changes in LDL particle size, metabolic syndrome, and emerging cardiometabolic risk factors after a 6-week intervention. Twenty one men and postmenopausal women (mean+/-SD; age: 59.2+/-9.5 y, body weight: 89.2+/-16.8 kg, % body fat: 38.5+/-6.2%, body mass index: 29.52+/-3.03 kg/m 2) who had successfully lowered LDL-C with a statin (<130 mg/dl) were matched on sex, age, lipids, and type of statin and randomized to a very low carbohydrate ketogenic diet (LCD) or a low carbohydrate diet with increased physical activity (LCD+PA). Subjects received weekly dietary counseling from a registered dietitian and they were instructed to maintain body weight. Fasting blood and physiological measurements were taken at baseline, week 3, and week 6. LDL size was measured with non-gradient polyacrylamide gel electrophoresis and vertical auto profile ultracentrifugation. Food records (mean+/-SD; baseline: 2159+/-415 kcals, 45/36/20 %carb/fat/pro; intervention: 1739+/-481 kcals, 11/58/26 %carb/fat/pro), urine acetoacetic acid, and serum total ketones demonstrated dietary compliance. LCD+PA incrementally increased average daily steps using a pedometer, reaching 4,792 steps greater than baseline at week 6. LDL particle size (p≤0.05) and reactive hyperemia peak forearm blood flow significantly increased (p≤0.01) and TAG (-36%), insulin (-25%), systolic (-5%) and diastolic (-6%) blood pressure, body weight (-3%), and waist circumference (-4%) decreased. Change in body weight was not significantly correlated with changes in blood markers. There were no significant differences observed for total cholesterol, LDL-C, HDL-C, Lp(a), glucose, or flow-mediated dilation, nor were group differences found. These findings indicate that individuals on statin medication showing signs of metabolic syndrome respond favorably to a low |
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Cholesterol Revitaliser $13.95 About one in every three people in the US, UK and Europe have elevated cholesterol. So there is a very good chance that you have raised cholesterol and may not even know it.It is a silent killer, and you need to find out NOW how to combat it.”Cholesterol Revitaliser” is a fast-track health solution that can help you to do exactly that. It covers everything you need to know about effectively lowering your cholesterol.It explores the information that you need to make informed, intelligent decisions about your health.It discusses natural ways that you can dramatically lower your cholesterol, as well as the role that statins and supplements can play. It outlines diet choices to lower cholesterol naturally and explores the amazing variety of foods that can re-invigorate your system.Cholesterol Revitaliser covers everything that you need to know about effectively lowering your cholesterol, revitalising your health and cutting your chances of getting heart disease and heart attacks. Dramatically.Throughout Cholesterol Revitaliser you will find hard facts backed up by scientific studies. Facts. Not theory. In language that you can actually understand.By the end you will know these 7 things that you have probably never heard anywhere else before:1/ The Fat Lowdown… And How Swapping 30 Calories of One Dietary Substance for Another DOUBLES Your Risk of Heart Disease! (If you know what it is you can avoid it)2/ 110,000 People Can’t Be Wrong… The SHOCKING Statistic that could cut your risk of heart attack by 30%!3/ A ‘Food’ That Is 32% Fat, and Yet LOWERS YOUR CHOLESTEROL! (I Delve Into the Little Known Study That Tells All)4/ A Treatment That Lowers LDL Cholesterol by 8.7% in MONTH 1 and 15% by Month 4 and Makes HDL Cholesterol Rise by 10%! (And it doesn’t have the side effects of many commercial treatments!)5/ Another ‘Natural’ Treatment that could reduce your LDL Cholesterol by 10 to 20%. Reduce Triglyceride’s by 20 to 50% |
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Cholesterol: The Good, the Bad, and the Ugly and What That Can Mean For You $6.99 Understanding cholesterol and how it affects the body can be a tricky subject, since there are two different types of cholesterol. Generally, when we talk about having high cholesterol, we mean the “bad’ kind, commonly called LDL cholesterol. The real trick is learning how to lower cholesterol and keep it low to prevent heart attacks and strokes.You may be asking yourself “exactly what is cholesterol?”Cholesterol is something that we need in the body. This substance is a soft, waxy fat found in the blood stream and used in the production of cell membrane and hormones. Without some amount of cholesterol in our bodies, we would die. However, sufficient cholesterol is produced in the liver, and so it is rarely necessary that we need to ingest extra amounts of this substance in order to live. We need both a measure of high cholesterol and low cholesterol, also known as LDL and HDL. In fact, low cholesterol is not any better than high cholesterol when it comes to our health.The fact is, most of us that battle too much cholesterol is in the area of having too much.If you are looking for ways of lowering cholesterol, you have come to the right place!If you want to recognize foods high in cholesterol, we can help!Once you have a copy of “Cholesterol: The Good, the Bad, and the Ugly and What That Can Mean For You” in your possession, you will learn things such as:•Cholesterol normal levels and what that means for you•Foods to lower cholesterol and where you can find them•Lowering cholesterol naturally and still enjoy good food•Lowering cholesterol with diet and not starving yourself to deathAnd much more!Contained inside this powerful eBook is everything you need to know in order to effectively diagnose and then treat high cholesterol. Nothing is ignored. Everything is covered. The fear and uncertainty of cholesterol will be |
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Coconut Oil for Life: Discover How Coconut Oil Can Boost Metabolism, Stimulate Thyroid Function, Improve Cholesterol, Help Curb Sugar Cravings and Aid in Weight Loss $17.95 Coconut Oil for Life: Over 250 Classic Recipes Reworked with Coconut Oil, by DJ Ley, is a condensed primer to introduce her readers to the miraculous health benefits of cooking with coconut oil. Discover how coconut oil can boost metabolism, stimulate thyroid function, lower LDL cholesterol while raising HDL cholesterol levels, help curb sugar cravings and aid your weight loss efforts. Through years of research, Ley found the answers to an over-prescribed and undernourished loved one with deteriorating health. By using the recipes and techniques in this book, DJ began preparing meals which stopped the downward spiral of her husband disease, gave him new vitality and repaired their marriage.Ley has developed luscious recipes presented in easily understood categories such as Main Meals, Vegetarian Dishes, Seafood, Chicken and Red Meat entrees, Dessert categories such as Cakes, Cookies and Guilt-Free Coconut Creations, Salad creations with Dressings, Dips, Spreads and Sauces, Breakfast Foods and Luncheon categories such as Pizza, a wide variety of Sandwiches, Soups and Salads, all presented with easy-to-follow instructions designed for the beginner as well as the experienced cook alike. To complete the selections, the book even has a section of Healthy Tidbits, Smoothies and Coconut Snacks to satisfy the cravings for those tempted to snack between meals. Finally, Ley supplies a poven Seven Day Diet Detox.This title is a must for those health conscious cooks who wish to serve nutritional, yet tasty meals for your family and friends while benefiting from the unique properties of coconut oil.Coconut Oil For Life: Discover How Coconut Oil Can Boost Metabolism, Stimulate Thyroid function, Improve Cholesterol, Help Curb Sugar Cravings and Aid in Weight Loss. Includes a 7 Day Detox Plan. |
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Determinants of cholesterol and lipoprotein metabolism as influenced by dietary fatty acid profile and cholesterol status in the F1B golden-Syrian hamster. $49.99 Investigated first was the effect of cholesterol status and dietary fatty acid profile on the expression of genes regulating hepatic cholesterol and lipoprotein metabolism. F1B hamsters were fed for 12 weeks diets enriched in 10% (w/w) coconut, olive or safflower oil in combination with either 0.1% cholesterol (cholesterol-supplemented) or 0.01% cholesterol (plus 10 days prior to killing 0.15% lovastatin and 2% cholestyramine) (cholesterol-depleted). Cholesterol depletion resulted in significantly lower plasma non-high density lipoprotein (HDL) cholesterol, HDL cholesterol and triglyceride concentrations. This was associated with up-regulation of genes involved in cholesterol uptake and excretion, and reverse cholesterol transport, and down-regulation of genes involved in de novo lipoprotein and cholesterol synthesis. Coconut oil fed hamsters had significantly higher non-HDL cholesterol and triglyceride concentrations than olive and safflower oil fed hamsters but was not associated with changes in the expression of genes regulating cholesterol and lipoprotein metabolism. Next, the mechanisms by which n-3 PUFA, relative to n-6 PUFA, alter cholesterol and lipoprotein metabolism were assessed using the same hamster model as described. Cholesterol-supplemented hamsters fed fish oil, relative to safflower oil, had higher plasma non-HDL cholesterol and triglyceride concentrations which was associated with down-regulation of genes involved in hepatic cholesterol uptake, and triglyceride and cholesteryl ester synthesis, and up-regulation of genes involved in de novo lipoprotein synthesis. In contrast, cholesterol-depleted hamsters fed fish oil, relative to safflower oil, had lower plasma non-HDL cholesterol and triglyceride concentrations which was associated with lower hepatic SREBP-1 mRNA and protein levels. Independent of cholesterol status, hamsters fed fish oil, relative to safflower oil, had lower HDL cholesterol concentrations and was associated with lower hepatic |
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Determinants of cholesterol and lipoprotein metabolism as influenced by dietary fatty acid profile and cholesterol status in the F1B golden-Syrian hamster. $49.99 Investigated first was the effect of cholesterol status and dietary fatty acid profile on the expression of genes regulating hepatic cholesterol and lipoprotein metabolism. F1B hamsters were fed for 12 weeks diets enriched in 10% (w/w) coconut, olive or safflower oil in combination with either 0.1% cholesterol (cholesterol-supplemented) or 0.01% cholesterol (plus 10 days prior to killing 0.15% lovastatin and 2% cholestyramine) (cholesterol-depleted). Cholesterol depletion resulted in significantly lower plasma non-high density lipoprotein (HDL) cholesterol, HDL cholesterol and triglyceride concentrations. This was associated with up-regulation of genes involved in cholesterol uptake and excretion, and reverse cholesterol transport, and down-regulation of genes involved in de novo lipoprotein and cholesterol synthesis. Coconut oil fed hamsters had significantly higher non-HDL cholesterol and triglyceride concentrations than olive and safflower oil fed hamsters but was not associated with changes in the expression of genes regulating cholesterol and lipoprotein metabolism. Next, the mechanisms by which n-3 PUFA, relative to n-6 PUFA, alter cholesterol and lipoprotein metabolism were assessed using the same hamster model as described. Cholesterol-supplemented hamsters fed fish oil, relative to safflower oil, had higher plasma non-HDL cholesterol and triglyceride concentrations which was associated with down-regulation of genes involved in hepatic cholesterol uptake, and triglyceride and cholesteryl ester synthesis, and up-regulation of genes involved in de novo lipoprotein synthesis. In contrast, cholesterol-depleted hamsters fed fish oil, relative to safflower oil, had lower plasma non-HDL cholesterol and triglyceride concentrations which was associated with lower hepatic SREBP-1 mRNA and protein levels. Independent of cholesterol status, hamsters fed fish oil, relative to safflower oil, had lower HDL cholesterol concentrations and was associated with lower hepatic |
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Effect Of Wheat Germ (Triticum Sativum) On The Hyperlipidemic Subjects $78.99 The study is clinical cum supplementation study. The study was conducted in the urban area of Puducherry. Purposive sampling method using snow ball technique was adapted to select 60 hyperlipidemic subjects from 93 samples between the age 40 and 60 years residing in the urban limits of Puducherry.Among the selected subjects, 30 subjects served as experimental group and the other 30 subjects formed the control group.The experimental groups were supplemented with 20g of wheat germ every day for a period of 90 days along with the regular diet.The findings in the study indicate that 20g per day of wheat germ for 90 days had a significant effect in lowering fasting blood sugar levels and post prandial blood sugar levels of the diabetic- hyperlipidemic subjects and total cholesterol levels, triglyceride levels, LDL cholesterol levels, VLDL cholesterol levels, total cholesterol/HDL cholesterol ratio, LDL cholesterol/HDL cholesterol ratio and fat percentage of the hyperlipidemic subjects.The study therefore confirms that the incorporation of wheat germ in the daily diet of the hyperlipidemic subjects could be beneficial. |
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Effect of carbohydrate restriction and American Heart Association diets on the clinical features of the metabolic syndrome, the inflammatory response and lipoprotein metabolism in young Emirati adults. $49.99 The incidence of diabetes mellitus (DM) in the UAE is one of the highest world-wide. The metabolic syndrome (MetS) characterized by central obesity, high blood pressure and dyslipidemias predisposes for the development of DM. The presence of MetS was assessed in a population of overweight/obese individuals by randomly screening 227 subjects, 18-50 (30.2 +/- 8.4 y), from Al-Ain city, Emirate of Abu Dhabi. 92 subjects (40.5%) were identified as having MetS. The most relevant clinical criteria associated with MetS were large waist circumference (WC), high systolic blood pressure and low HDL cholesterol (HDL-C).;Dietary interventions were conducted in 39 subjects. Initially, all individuals followed a carbohydrate restricted diet (CRD) with 25% energy from carbohydrate. After 6 wk, half of the subjects (n=19) were switched to the low fat diet (55% energy from carbohydrate) recommended by the American Heart Association (AHA group) while the other half (n=20) continued with the CRD diet for additional 6 wk (CRD group). Diet records indicated high compliance with the dietary guidelines. At wk 6, all subjects presented decreases in body weight (P < 0.0001), WC (P < 0.001), body fat (P < 0.0001) and plasma triglycerides (TG) (P < 0.0001). Significant decreases were also seen in plasma LDL cholesterol, blood pressure, insulin and inflammation markers while a significant increase in adiponectin was observed. After 12 wk, these positive changes persisted for all subjects independent of diet. However, body weight, plasma TG, insulin and plasma glucose were lower in the CRD (P < 0.05) compared to the AHA group.;The large atherogenic VLDL subfraction was decreased over time for all subjects (P < 0.01) with a more pronounced decrease (P <0.05) in the CRD compared to the AHA group. Medium and small LDL particles and apolipoprotein B decreased for all subjects (P < 0.01) rendering a less atherogenic lipoprotein profile. These studies indicate that CRD can |
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Effect of carbohydrate restriction and American Heart Association diets on the clinical features of the metabolic syndrome, the inflammatory response and lipoprotein metabolism in young Emirati adults. $49.99 The incidence of diabetes mellitus (DM) in the UAE is one of the highest world-wide. The metabolic syndrome (MetS) characterized by central obesity, high blood pressure and dyslipidemias predisposes for the development of DM. The presence of MetS was assessed in a population of overweight/obese individuals by randomly screening 227 subjects, 18-50 (30.2 +/- 8.4 y), from Al-Ain city, Emirate of Abu Dhabi. 92 subjects (40.5%) were identified as having MetS. The most relevant clinical criteria associated with MetS were large waist circumference (WC), high systolic blood pressure and low HDL cholesterol (HDL-C).;Dietary interventions were conducted in 39 subjects. Initially, all individuals followed a carbohydrate restricted diet (CRD) with 25% energy from carbohydrate. After 6 wk, half of the subjects (n=19) were switched to the low fat diet (55% energy from carbohydrate) recommended by the American Heart Association (AHA group) while the other half (n=20) continued with the CRD diet for additional 6 wk (CRD group). Diet records indicated high compliance with the dietary guidelines. At wk 6, all subjects presented decreases in body weight (P < 0.0001), WC (P < 0.001), body fat (P < 0.0001) and plasma triglycerides (TG) (P < 0.0001). Significant decreases were also seen in plasma LDL cholesterol, blood pressure, insulin and inflammation markers while a significant increase in adiponectin was observed. After 12 wk, these positive changes persisted for all subjects independent of diet. However, body weight, plasma TG, insulin and plasma glucose were lower in the CRD (P < 0.05) compared to the AHA group.;The large atherogenic VLDL subfraction was decreased over time for all subjects (P < 0.01) with a more pronounced decrease (P <0.05) in the CRD compared to the AHA group. Medium and small LDL particles and apolipoprotein B decreased for all subjects (P < 0.01) rendering a less atherogenic lipoprotein profile. These studies indicate that CRD can |
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Effect of varying the fatty acid composition of a carbohydrate-restricted diet on plasma fatty acid composition, blood lipids oxidative stress, inflammation and insulin sensitivity in men. $49.99 Background. Carbohydrate-restricted diets (CRD) consistently improve risk factors associated with Metabolic Syndrome (MetSyn). Recently, we showed that a saturated-fat rich hypocaloric CRD significantly reduced serum content of saturated fatty acids (SFA) and significantly increased arachidonic acid (AA) and the omega-6/omega-3 polyunsaturated (PUFA) ratio while significantly decreasing inflammation compared to a low-fat diet. This disconnect between dietary and circulating lipid lead us to explore how varying the fat composition of a CRD affects these variables, in addition to oxidative stress. Methods. Eight healthy weight-stable men (age 45 +/- 7.9 y, body fat 28.4 +/- 6.5%) were fed two eucaloric CRD varying in saturated fat and unsaturated fat with the same macronutrient distribution (12%en carbohydrate, 30%en protein, 58%en fat, 850 mg cholesterol) for 6 weeks each without weight loss. Similar foods were fed but CRD-SFA emphasized dairy fat while CRD-UFA emphasized olive oil, omega-3 fortified eggs, salmon, and walnuts. CRD-SFA provided almost twice as much SFA (86 g vs 47 g) and less monounsaturated fat (MUFA) and omega-6 and omega-3 PUFA than CRD-UFA, confirmed by chemical analysis. Fasting blood and 24 hr urine was analyzed at baseline and following each diet for fasting lipoproteins, insulin, glucose, inflammatory markers, fatty acid composition in plasma triacylglycerides (TAG), phospholipids (PL) and cholesteryl esters (CE), and urine 8-iso PGF2alpha. Results. Regardless of fat quality, both CRD significantly decreased TAG and insulin, and increased HDL-C and LDL particle size (P < 0.05). Despite increased total-cholesterol (TC) and LDL-cholesterol (LDL-C) after CRD-SFA, the TC/LDL-C ratio was not different between diets. Total plasma SFA in CE, PL, and TAG were unchanged after the CRD-SFA, and plasma PL and CE AA content was significantly increased. CRD-UFA significantly increased PL and CE EPA+DHA content and the PL omega-3 index. Inflammation was |
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Effect of varying the fatty acid composition of a carbohydrate-restricted diet on plasma fatty acid composition, blood lipids oxidative stress, inflammation and insulin sensitivity in men. $49.99 Background. Carbohydrate-restricted diets (CRD) consistently improve risk factors associated with Metabolic Syndrome (MetSyn). Recently, we showed that a saturated-fat rich hypocaloric CRD significantly reduced serum content of saturated fatty acids (SFA) and significantly increased arachidonic acid (AA) and the omega-6/omega-3 polyunsaturated (PUFA) ratio while significantly decreasing inflammation compared to a low-fat diet. This disconnect between dietary and circulating lipid lead us to explore how varying the fat composition of a CRD affects these variables, in addition to oxidative stress. Methods. Eight healthy weight-stable men (age 45 +/- 7.9 y, body fat 28.4 +/- 6.5%) were fed two eucaloric CRD varying in saturated fat and unsaturated fat with the same macronutrient distribution (12%en carbohydrate, 30%en protein, 58%en fat, 850 mg cholesterol) for 6 weeks each without weight loss. Similar foods were fed but CRD-SFA emphasized dairy fat while CRD-UFA emphasized olive oil, omega-3 fortified eggs, salmon, and walnuts. CRD-SFA provided almost twice as much SFA (86 g vs 47 g) and less monounsaturated fat (MUFA) and omega-6 and omega-3 PUFA than CRD-UFA, confirmed by chemical analysis. Fasting blood and 24 hr urine was analyzed at baseline and following each diet for fasting lipoproteins, insulin, glucose, inflammatory markers, fatty acid composition in plasma triacylglycerides (TAG), phospholipids (PL) and cholesteryl esters (CE), and urine 8-iso PGF2alpha. Results. Regardless of fat quality, both CRD significantly decreased TAG and insulin, and increased HDL-C and LDL particle size (P < 0.05). Despite increased total-cholesterol (TC) and LDL-cholesterol (LDL-C) after CRD-SFA, the TC/LDL-C ratio was not different between diets. Total plasma SFA in CE, PL, and TAG were unchanged after the CRD-SFA, and plasma PL and CE AA content was significantly increased. CRD-UFA significantly increased PL and CE EPA+DHA content and the PL omega-3 index. Inflammation was |
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Fenofibrate $43.04 High Quality Content by WIKIPEDIA articles! Fenofibrate is a drug of the fibrate class. Fenofibrate was discovered by Groupe Fournier SA, before it was acquired in 2005 by Solvay Pharmaceutical, a business unit owned by the Belgian corporation, Solvay S.A. It is mainly used to reduce cholesterol levels in patients at risk of cardiovascular disease. Like other fibrates, it reduces both low-density lipoprotein (LDL) and very low density lipoprotein (VLDL) levels, as well as increasing high-density lipoprotein (HDL) levels and reducing tryglycerides level. |
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Food Cures: Fight Disease with Your Fork! $17.95 Clean out your medicine cabinet and restock with healing and appealing items from the grocery store. Treat the most common ailments naturally, safely, and deliciously-and live pain free, disease free, and worry free! Rely less on pills and more on real food. How much? How often? Here you’ll find all the answers, the research-validated treatments, and successful cures for dozens of common conditions. The past few years have been filled with intriguing announcements from the world of medical research. Forget about wonder drugs-we’re living in a time of wonder foods. The foods described in this book are nutritional powerhouses bursting with compounds that have specific and well-defined health benefits-included are 75 healthy recipes that will please every member of the family. Healing foods section includes information about:• How a rainbow of fruits and vegetables (8 to 9 servings a day)-the wider the variety the better-will lower the risk of an array of cancers• How dark chocolate contains hefty amounts of disease-fighting flavonoids and can significantly improve blood pressure• How olive oil lowers “bad” LDL cholesterol and raises “good” HDL cholesterol• How spinach, in addition to protecting your eyes from macular degeneration, is a valuable source of vitamin K, which can help maintain bone densityDiscover cures for common conditions from allergies to gum disease, from insomnia to lowering your cholesterol. Changing your diet won’t guarantee that you’ll never get sick or need drugs, but eating the right food can help heal what ails you and can bolster your body’s defenses against disease, treat disease directly, and even slow the aging process. |
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Good Cholesterol, Bad Cholesterol: An Indispensable Guide to The Facts About Cholesterol $9.95 High cholesterol is an important risk factor for heart disease, heart attack, and strokes. And although cholesterol screening is one of the most widely administered and evaluated blood tests, many people don’t realize that our bodies require some cholesterol to function normally and remain confused about which cholesterol is the “good” one and which is the “bad.” Now, in Good Cholesterol, Bad Cholesterol, Hirsch presents a clear, concise primer to all the varieties of this blood lipid, including HDL (high-density lipoprotein), LDL (low-density lipoprotein), VDL (very low density lipoprotein), and others, as well as the connection between the ways various types of fat affect cholesterol levels. Providing essential information about how cholesterol relates to heart disease, stroke, and other life-threatening medical conditions, this essential primer also provides guidance about how to manage your cholesterol levels—through diet, exercise, and stress management—for optimum, lifelong health. |
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Guggul: Ayurveda’s Wonder Herb $1.99 Guggul (Commiphora mukul), long used by ayurvedic practitioners, possesses strong rejuvenating and purifying qualities. A potent antioxidant, guggul acts to lower “bad” LDL cholesterol levels and raise “good” HDL cholesterol levels. In addition, guggul has strong anti-inflammatory properties that can be useful in treating arthritis and other chronic inflammatory conditions. |
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High-Density Lipoproteins: From Basic Biology to Clinical Aspects $235 This book meets the long-awaited need for a comprehensive overview of the biological role of HDLs. Edited by one of the pioneers of HDL and cholesterol research, and with major contributions by leading HDL researchers and clinicians from around the world, this monograph summarizes current knowledge on HDL turnover, regulation and physiology. Clearly structured, the various sections cover HDL structure, formation, secretion and removal, as well as plasma metabolic factors. The biological activities and clinical aspects are fully discussed, as is the impact of HDL on common diseases and their prevention.From the contents:HDL StructureHDL Plasma Metabolic FactorsHDL Formation, Secretion, RemovalBiological Activities of HDLClinical Aspects of HDL |
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How to Get in Shape Fast! $4.99 Want to learn how to get in shape fast? Looking for the best way to get in shape? Desirous to get in shape for summer? Maybe it’s time you picked up a copy of How to Get in the Shape Fast! and start learning ways to get in shape and get in shape fast.If you have no time in your day . . . If you don’t know the difference between a barbell and a cowbell . . . You can find a way to add simple, quick exercises to your daily regimens for a slimmer waistline, increased energy, and a happier life!It can be hard to get to the gym after a long day at work. Your family expects you back as soon as possible. You’re pent up in a cubicle all day, never able to stretch. Or constantly leaving for business trips.Anyone who’s ever been in your shoes knows it can be difficult to keep a regular workout routine.The truth is, you can get exercise without working out. You can now get in shape fast!You’re about to discover how you can fit simple, quick exercises into your day and get in shape quick…even if you have to multi-task.You can melt away those extra pounds, increase your energy and feel good about your health.Learn all the tips, tricks, and tactics of how to get in shape quick from those who have to be creative to stay active. You’re about to discover :*A new way to look at how you treat and care for your body*5 ways people end up looking older than they really are and how you can avoid it*3 questions you must ask yourself to honestly judge how fit you are*The secret behind “slowly but surely” and how it can save the day for people on the go*5 reasons you’ll never regret getting in shape*The lowdown on HDL/LDL counts, your cholesterol, and the risks of being in the red zone*If you’re a woman, exercise can be one of your greatest weapons against osteoporosis*How to prevent diabetes with exercise*How being |
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Magic Herbs: More Than 200 Delicious & Healthy Recipes That are Naturally Low-Fat & Fat-Free $19.95 A natural remedy for monotonous meals. In ancient times herbs were thought to heal and perform other wonders. While their magic powers may be just folklore, herbs do provide health benefits—like regulating blood sugar and appetite and raising levels of "good" (HDL) cholesterol. But the real magic of herbs lies in the distinctive flavors they add to foods. They transform low-fat, low-salt meals from, dull to delicious. Magic Herbs is a collection of over 200 recipes that make it easy to spice up your diet, using more than 50 types of herbs in savory dishes that are naturally low-fat and fat-free. Mouthwatering creations include:Arugula and Turkey Stuffed MushroomsDill Weed and Thyme Whole Wheat Herb BreadGrilled Porterhouse Steaks and Basil TomatoesBay Leaf and Cayenne Shrimp CreoleNutmeg Orange Marinated ChickenEasy Chive and Garlic Twice-Baked PotatoesCinnamon Pumpkin TorteBergamot Strawberry Iced TeaEach easy-to-make recipe provides preparation time and complete nutrition analysis and exchange information. This unique book also features a "dictionary" of herbs, tips on growing and drying your own, and suggestions for using herbs in decorating and gift-giving. |
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My Baby Boomer Baby Book: A Record of Milestones, Millstones & Gallstones $13.95 Congratulations, baby boomers: You are now officially all middle-aged. It’s a book of firsts: My first colonoscopy, my first reading glasses. A book of vital statistics, including married name(s), circumference of abdomen, cholesterol count (HDL and LDL), and home state (Red or Blue). It’s a place to keep track of primary care givers—herbalist, psychopharmacologist. Record favorite expressions—I’m having a senior moment. Dressing on the side, please. 60 is the new 50. Keep track of “What I’ve Grown,” from liver spots to knee flaps. There’s also a place for a lock of hair (if you can spare it) along with the Seven Stages of Hair Loss (men: from minoxidil to shaves head; women: from plucks grey hairs to dyes it champagne blond). Plus essays—”Am I Smiling or Is It Gas,” and “I Go to School,” a parody of Adult Ed classes. |
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Nutritional composition and hypocholesterolemic effect of Mushroom $61.99 Mushroom cultivation has been started recently in Bangladesh. People in Bangladesh are still not very aware of the nutritional and medicinal importance of mushrooms. In this study, the nutritional values of two species of mushrooms (Pleurotus sajor-caju and P. florida) have been determined. These mushrooms are rich in proteins and fibers, and contain a lower amount of lipid. They are also rich in mineral contents. The feeding of hypercholesterolemic rats with 5% powder of fruiting bodies of P. sajor-caju and P. florida reduced the plasma total cholesterol, triglyceride, LDL/HDL ratio level significantly. These properties may be due to several mechanisms, one of which is the increased cholesterol excretion through feces by mushroom feeding. Dietary mushroom P. florida also reduced lipid peroxidation in hypercholesterolemic rats, this data suggests that this mushroom might be an important antioxidant. This study revealed that P. sajor-caju and P. florida mushrooms are nutritionally valuable food items with high levels of protein and fiber. And mushrooms might be good nutrition source for heart patients and also for diabetic and cancer patients. |
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Overcoming Metabolic Syndrome $19.95 Twenty-seven Percent of Americans Have Metabolic Syndrome It’s now estimated that 55 million Americans have metabolic syndrome; however, if you’re like most Americans, you may not have ever heard of the syndrome much less understand how it increases your risk of heart disease and stroke. Metabolic syndrome actually is a cluster of disorders, all of which are affected by both genetics and lifestyle. If you have three or more of the following, you considered as having metabolic syndrome:     ·        A waistline of 40 inches or more for men and 35 inches or more for women·        High triglyceride levels·        High blood pressure·        Elevated fasting blood  sugar levels·        Low HDL cholesterol levels (the good cholesterol)  There are no specific symptoms of metabolic syndrome, but it takes a toll on health. People with metabolic syndrome are 3.5 times more likely to die of a heart-related ailment and 5 times more likely to develop type 2 diabetes. The good news is that you can overcome this deadly syndrome. The authors of Overcoming Metabolic Syndrome, an endocrinologist and a cardiovascular surgeon, have treated thousands of patients; they know how to cut through the confusion and give you’re the basics about how you can restore your health. Among the questions they answer:·        How do I know if I have metabolic syndrome?·        What is insulin resistance? Why is it a critical issue?·        Why does insulin resistance make it so hard to lose |
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Phospholipid Transfer Protein and Cholesteryl Ester Transfer Protein $59 Cardiovascular diseases are an important cause of mortality worldwide. They are an entity of diseases of the heart and vasculature, mainly caused by atherosclerosis. In atherosclerosis, cholesterol accumulates in the vessel wall, mainly in the form of low-density lipoprotein (LDL). High-density lipoprotein (HDL) protects against atherosclerosis, as HDL particles accept cellular cholesterol and transport it to the liver for excretion in a process called reverse cholesterol transport (RCT). Phospholipid transfer protein (PLTP) and cholesteryl ester transfer protein (CETP) are two key lipid transfer proteins responsible for the remodeling of HDL particles. The aim of this thesis was to clarify the role of PLTP and CETP in RCT. The results indicate that both PLTP and CETP can remodel HDL as a cholesterol acceptor, which affects the removal of cholesterol from foam cells. This thesis is useful to anyone interested in lipid metabolism. It gives the reader a basic knowledge about the development of atherosclerosis and factors affecting that. It is also useful to professionals giving also a deeper insight to the world of lipid metabolism. |
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Pulse wave analysis in type 1 diabetes: Relationship with historical measures and prevalent disease. $49.99 Type 1 diabetes (T1D) is associated with numerous complications. These include renal and cardiovascular disease which are the leading causes of morbidity and mortality in T1D. Renal complications also increase the risk for cardiovascular disease. Early detection and treatment of their risk factors may help to prevent or at least delay these complications. This dissertation examines potential risk factors for altered measures of pulse wave analysis (PWA), which have been linked to cardiovascular events and mortality in other populations. It also examines how PWA measures relate to prevalent cardiovascular and renal complications in T1D.;Prospective analyses of potential risk factors for increased arterial stiffness indices, augmentation index (AIx) and augmentation pressure (AP), and decreased estimated myocardial perfusion, i.e. subendocardial viability ratio (SEVR), showed autonomic neuropathy, smoking history, low HDL cholesterol and poorer glycemic control, to be associated with altered PWA measures 18 years later.;Next, cross-sectional analyses between PWA measures and prevalent CVD showed AP and SEVR to be significantly related to coronary artery disease and coronary artery calcification, respectively, although age was the major predictor of both. AP was also higher, although not significantly, and SEVR significantly lower in those with peripheral vascular disease.;Finally, SEVR, but not AIx nor AP, was significantly associated with the presence of microalbuminuria (MA), and preferentially entered multivariate models over brachial blood pressure measures. SEVR was also related to degree of albuminuria in those within the normo- and MA range, and was significantly associated, multivariately, with low renal function.;This dissertation thus yields significant Public Health findings by identifying factors (AN, smoking, glycemic control, lipid levels) that may delay increased arterial stiffness (AIx and AP) and decreased myocardial perfusion (SEVR). As it |
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Qadir’s Test $49.99 Please note that the content of this book primarily consists of articles available from Wikipedia or other free sources online. High Quality Content by WIKIPEDIA articles! Qadir’s test is used for the diagnosis of cancer. The plasma lipid profile is changed in cancer patients. So the change in plasma lipid profile may be used as marker for diagnosis of cancer. The test was named by a scientist Dr. M.I. Qadir who worked on it. Lipids are carried in body fluids with the help of lipoproteins, chylomicrons transport of triglycerides from the intestine to all cells. Very low-density lipoproteins (VLDL) are involved in the transportation of triglycerides from the liver to other cells. Low-density lipoproteins (LDL) are responsible for the transport of cholesterol from liver to the cells and high density lipoproteins (HDL) are involved for the transport of cholesterol from cells to the liver. Chylomicrons and very low density lipoproteins are rapidly catabolized. Thus triglycerides, cholesterol, LDL-cholesterol and HDL-cholesterol constitute Plasma Lipid Profile. |
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The HDL Handbook: Biological Functions and Clinical Implications $149.95 The HDL Handbook: Biological Functions to Clinical Implications brings laboratory research in HDL from bench to bedside in this needed resource for researchers and clinicians studying cholesterol, lipids, epidemiology, biochemistry, molecular medicine, and pathophysiology of cardiovascular diseases. In addition researchers and clinicians working with an aging population, corporate researchers, postdocs; medical students and graduate students will find this publication useful, as the scope of coverage includes basic science, genetics, epidemiology, treatment of HDL cholesterol, as well as, potential targets to modify HDL cholesterol. Comprehensive coverage of basic science, genetics, epidemiology and treatment Reputable content on latest advances in HDL cholesterol research Inclusive, worldwide content with country specific information In depth discussion of potential targets to modify HDL |
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The HDL Handbook: Biological Functions and Clinical Implications $106.49 The HDL Handbook: Biological Functions to Clinical Implications brings laboratory research in HDL from bench to bedside in this needed resource for researchers and clinicians studying cholesterol, lipids, epidemiology, biochemistry, molecular medicine, and pathophysiology of cardiovascular diseases. In addition researchers and clinicians working with an aging population, corporate researchers, postdocs; medical students and graduate students will find this publication useful, as the scope of coverage includes basic science, genetics, epidemiology, treatment of HDL cholesterol, as well as, potential targets to modify HDL cholesterol. Comprehensive coverage of basic science, genetics, epidemiology and treatment Reputable content on latest advances in HDL cholesterol research Inclusive, worldwide content with country specific information In depth discussion of potential targets to modify HDL |
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The Natural Guide to Managing Prediabetes $0.4 Are you concerned that you may be diagnosed with diabetes? Does diabetes run in your family? Are you overweight? Do you have high blood pressure? Do you have high LDL cholesterol and low HDL cholesterol levels? Does your blood glucose level tend to be in the high-normal range? Have you had gestational diabetes? If you can answer yes to any two of these questions then you may have pre-diabetes, metabolic syndrome, or Syndrome X – three terms that signify you as a good candidate to be diagnosed with diabetes at some point. In this booklet, Martin Stone outlines the things you can do now – such as eating a proper diet, getting adequate exercise, and taking the right herbs and nutritional supplements-to avoid the sixth most common cause of death in the United States. |
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The Tubby Theory From Topeka $19.99 It is time for Americans and their physicians to learn that there is more to do to prevent heart attacks and strokes.Everyone must learn the difference between LDL-C and LDL-P and non-HDL cholesterol (The Tubby Factor).Everyone must learn what a calcium score and a carotid intima thickness ultrasound does to detect subclinical atherosclerosis.My book, The Tubby Theory from Topeka teaches the above and demonstrates how I used the above tests to prevent heart disease and stroke and to also regress plaque in the arteries in my medical practice in Topeka for the last two years.My goal is to change the Paradigm of preventive medicine in America. The new paradigm is to find subclinical atherosclerosis early with a calcium score and a CIMT to then treat it to stabilize the vulnerable plaque from inflammation and rupture.Rupture of the plaque causes sudden death.In 1 of 3 patients, SUDDEN DEATH is the first sign of heart disease.Reference: Lipoprotein Management in Patients with Cardiometabolic Risk, Brunzell et al, JACC. 2008; 51: 1513Tim Russert died one year ago of sudden death. The media reported nothing more could have been done for him. Non-HDL cholesterol goal of < 100 was not met. This was never reported. It was reported that his LDL-C was to goal 68 and his nuclear stress test was normal one month before his death.It has been more than a year since Mr. Russert died. I hope my new term that I coined, The Tubby Factor, will bring attention to the above facts. |
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The Tubby Theory From Topeka $9.99 It is time for Americans and their physicians to learn that there is more to do to prevent heart attacks and strokes.Everyone must learn the difference between LDL-C and LDL-P and non-HDL cholesterol (The Tubby Factor).Everyone must learn what a calcium score and a carotid intima thickness ultrasound does to detect subclinical atherosclerosis.My book, The Tubby Theory from Topeka teaches the above and demonstrates how I used the above tests to prevent heart disease and stroke and to also regress plaque in the arteries in my medical practice in Topeka for the last two years.My goal is to change the Paradigm of preventive medicine in America. The new paradigm is to find subclinical atherosclerosis early with a calcium score and a CIMT to then treat it to stabilize the vulnerable plaque from inflammation and rupture.Rupture of the plaque causes sudden death.In 1 of 3 patients, SUDDEN DEATH is the first sign of heart disease.Reference: Lipoprotein Management in Patients with Cardiometabolic Risk, Brunzell et al, JACC. 2008; 51: 1513Tim Russert died one year ago of sudden death. The media reported nothing more could have been done for him. Non-HDL cholesterol goal of < 100 was not met. This was never reported. It was reported that his LDL-C was to goal 68 and his nuclear stress test was normal one month before his death.It has been more than a year since Mr. Russert died. I hope my new term that I coined, The Tubby Factor, will bring attention to the above facts. |
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The effects of green tea supplementation in subjects with metabolic syndrome in Oklahoma. $49.99 Scope and method of study. Green tea, rich in flavonoids, has been shown to possess cardiovascular health benefits. This is a randomized controlled trial investigating whether green tea beverage or extract supplementation improved the cardiovascular risk profile associated with metabolic syndrome (MetS). Subjects with MetS were matched for age and sex and were randomly assigned to control (4 cups water/day), green tea (4 cups/day), or green tea supplement (2 capsules & 4 cups water/day) group for 8 weeks. Fasting blood samples, anthropometric measurements, and 3-day food records were taken at screening, 4 & 8 weeks. Blood samples were analyzed for lipid and glucose levels using standard clinical chemistry techniques. Dietary data were analyzed for nutritional content using Nutritionist Pro, version 3.2.;Findings and conclusions. Daily supplementation of decaffeinated green tea (4 cups/day) may be beneficial to humans diagnosed with the metabolic syndrome (MetS), as it may help decrease total body weight. Body weight and BMI decreased significantly (p<0.05) in green tea (-2.5 kg and -0.9, respectively) versus control (+0.6 kg and + 0.2 kg) at eight weeks. Although the supplement group did not reach significance, a similar weight loss trend was observed (-1.3 kg), which also decreased BMI (-0.4). When compared to controls, there were no significant differences in change at eight weeks for the green tea beverage or supplement group in fasting plasma glucose levels, lipid levels, body fat percentage, waist circumference, or blood pressure. However, a decrease in trend (p<0.1) of diastolic blood pressure at eight weeks for the green tea group (-4.7 mmHg) compared to placebo (+1.5 mmHg) was noted, and the same trend did not exist for the supplement group. An increase in trend (p<0.1) was observed for HDL cholesterol in green tea (+0.9 mg/dL) versus control (-1.4 mg/dL). A similar pattern was true for the supplement group (+0.6 mg/dL). At eight weeks, |
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The effects of green tea supplementation in subjects with metabolic syndrome in Oklahoma. $108 Scope and method of study. Green tea, rich in flavonoids, has been shown to possess cardiovascular health benefits. This is a randomized controlled trial investigating whether green tea beverage or extract supplementation improved the cardiovascular risk profile associated with metabolic syndrome (MetS). Subjects with MetS were matched for age and sex and were randomly assigned to control (4 cups water/day), green tea (4 cups/day), or green tea supplement (2 capsules & 4 cups water/day) group for 8 weeks. Fasting blood samples, anthropometric measurements, and 3-day food records were taken at screening, 4 & 8 weeks. Blood samples were analyzed for lipid and glucose levels using standard clinical chemistry techniques. Dietary data were analyzed for nutritional content using Nutritionist Pro, version 3.2.;Findings and conclusions. Daily supplementation of decaffeinated green tea (4 cups/day) may be beneficial to humans diagnosed with the metabolic syndrome (MetS), as it may help decrease total body weight. Body weight and BMI decreased significantly (p<0.05) in green tea (-2.5 kg and -0.9, respectively) versus control (+0.6 kg and + 0.2 kg) at eight weeks. Although the supplement group did not reach significance, a similar weight loss trend was observed (-1.3 kg), which also decreased BMI (-0.4). When compared to controls, there were no significant differences in change at eight weeks for the green tea beverage or supplement group in fasting plasma glucose levels, lipid levels, body fat percentage, waist circumference, or blood pressure. However, a decrease in trend (p<0.1) of diastolic blood pressure at eight weeks for the green tea group (-4.7 mmHg) compared to placebo (+1.5 mmHg) was noted, and the same trend did not exist for the supplement group. An increase in trend (p<0.1) was observed for HDL cholesterol in green tea (+0.9 mg/dL) versus control (-1.4 mg/dL). A similar pattern was true for the supplement group (+0.6 mg/dL). At eight weeks, |
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The effects of green tea supplementation in subjects with metabolic syndrome in Oklahoma. $49.99 Scope and method of study. Green tea, rich in flavonoids, has been shown to possess cardiovascular health benefits. This is a randomized controlled trial investigating whether green tea beverage or extract supplementation improved the cardiovascular risk profile associated with metabolic syndrome (MetS). Subjects with MetS were matched for age and sex and were randomly assigned to control (4 cups water/day), green tea (4 cups/day), or green tea supplement (2 capsules & 4 cups water/day) group for 8 weeks. Fasting blood samples, anthropometric measurements, and 3-day food records were taken at screening, 4 & 8 weeks. Blood samples were analyzed for lipid and glucose levels using standard clinical chemistry techniques. Dietary data were analyzed for nutritional content using Nutritionist Pro, version 3.2.;Findings and conclusions. Daily supplementation of decaffeinated green tea (4 cups/day) may be beneficial to humans diagnosed with the metabolic syndrome (MetS), as it may help decrease total body weight. Body weight and BMI decreased significantly (p<0.05) in green tea (-2.5 kg and -0.9, respectively) versus control (+0.6 kg and + 0.2 kg) at eight weeks. Although the supplement group did not reach significance, a similar weight loss trend was observed (-1.3 kg), which also decreased BMI (-0.4). When compared to controls, there were no significant differences in change at eight weeks for the green tea beverage or supplement group in fasting plasma glucose levels, lipid levels, body fat percentage, waist circumference, or blood pressure. However, a decrease in trend (p<0.1) of diastolic blood pressure at eight weeks for the green tea group (-4.7 mmHg) compared to placebo (+1.5 mmHg) was noted, and the same trend did not exist for the supplement group. An increase in trend (p<0.1) was observed for HDL cholesterol in green tea (+0.9 mg/dL) versus control (-1.4 mg/dL). A similar pattern was true for the supplement group (+0.6 mg/dL). At eight weeks, |
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The relationship between calcium intake, obesity, and cardiovascular disease risk factors: The Jackson Heart Study. $49.99 Cardiovascular disease (CVD) is a major health risk in the United States. Major indicators of CVD risk include obesity, blood lipids, and blood pressure. Modifiable risk factors associated with CVD include body composition (body mass index and waist circumference), serum lipids, and blood pressure. Data suggest calcium intake may play a role in regulation of weight, serum lipids, and blood pressure. The purpose of this study was to assess relationships of dietary calcium intake with weight status, and cardiovascular disease risks in African American population participating in the Jackson Heart Study. The subjects included 4,267 African American adults ages 21-95 years (mean = 55.1+/-12.6 years) in the Jackson Heart Study (JHS). Dependent variables included: body mass index (BMI) calculated from measured height/weight (stadiometer/balance scale), waist circumference (WC; measuring tape), serum lipids, and blood pressure (sphygmomanometer). A 158-item food frequency questionnaire (FFQ) was used to assess nutrient intake. Statistical analyses included multiple regression analysis and Pearson correlations using SPSS 16.0 (SPSS Inc. Chicago, IL, USA). There was a significant positive relationship between calcium intake and the body composition measure BMI [F (4, 3982) = 3.26, p = 0.019, DeltaR2 = .003] and a significant inverse relationship between calcium intake and WC [F (4, 3982) = 2.43, p = 0.05, DeltaR 2 = .002]. These relationships were also observed in females only when data were analyzed by gender. There were significant inverse relationships between calcium intake and total cholesterol (TC) [F (4, 4259 = 5.46, p = 0.266, DeltaR2 = .002] and LDL-cholesterol (LDL) [F (4, 4225) = 3.218 p = 0.01, DeltaR2 = .003]. There were also significant inverse correlations between total cholesterol (TC) and LDL and calcium for males only. There was a significant relationship between calcium intake and HDL-cholesterol [F (4, 4259) = 13.31, p < 0.001, DeltaR2 = .012], as |
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The relationship between calcium intake, obesity, and cardiovascular disease risk factors: The Jackson Heart Study. $49.99 Cardiovascular disease (CVD) is a major health risk in the United States. Major indicators of CVD risk include obesity, blood lipids, and blood pressure. Modifiable risk factors associated with CVD include body composition (body mass index and waist circumference), serum lipids, and blood pressure. Data suggest calcium intake may play a role in regulation of weight, serum lipids, and blood pressure. The purpose of this study was to assess relationships of dietary calcium intake with weight status, and cardiovascular disease risks in African American population participating in the Jackson Heart Study. The subjects included 4,267 African American adults ages 21-95 years (mean = 55.1+/-12.6 years) in the Jackson Heart Study (JHS). Dependent variables included: body mass index (BMI) calculated from measured height/weight (stadiometer/balance scale), waist circumference (WC; measuring tape), serum lipids, and blood pressure (sphygmomanometer). A 158-item food frequency questionnaire (FFQ) was used to assess nutrient intake. Statistical analyses included multiple regression analysis and Pearson correlations using SPSS 16.0 (SPSS Inc. Chicago, IL, USA). There was a significant positive relationship between calcium intake and the body composition measure BMI [F (4, 3982) = 3.26, p = 0.019, DeltaR2 = .003] and a significant inverse relationship between calcium intake and WC [F (4, 3982) = 2.43, p = 0.05, DeltaR 2 = .002]. These relationships were also observed in females only when data were analyzed by gender. There were significant inverse relationships between calcium intake and total cholesterol (TC) [F (4, 4259 = 5.46, p = 0.266, DeltaR2 = .002] and LDL-cholesterol (LDL) [F (4, 4225) = 3.218 p = 0.01, DeltaR2 = .003]. There were also significant inverse correlations between total cholesterol (TC) and LDL and calcium for males only. There was a significant relationship between calcium intake and HDL-cholesterol [F (4, 4259) = 13.31, p < 0.001, DeltaR2 = .012], as |
Category: Cholesterol Diet
